Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Filters

William Y Yang

Showing results (1-10 of 34) with videos related to

Pageof 4
Sort By:
Cytokine|June 27, 2017
IL-35, as a newly proposed homeostasis-associated molecular pattern, plays three major functions including anti-inflammatory initiator, effector, and blocker in cardiovascular diseasesXinyuan Li, Pu Fang, William Y Yang, et al.
Burns & Trauma|December 2, 2015
Pathological conditions re-shape physiological Tregs into pathological TregsWilliam Y Yang, Ying Shao, Jahaira Lopez-Pastrana, et al.
Redox Biology|November 16, 2019
Anti-inflammatory cytokines IL-35 and IL-10 block atherogenic lysophosphatidylcholine-induced, mitochondrial ROS-mediated innate immune activation, but spare innate immune memory signature in endothelial cellsXinyuan Li, Pu Fang, Yu Sun, et al.
Antioxidants & Redox Signaling|March 23, 2017
Lysophospholipids and Their Receptors Serve as Conditional DAMPs and DAMP Receptors in Tissue Oxidative and Inflammatory InjuryYing Shao, Gayani Nanayakkara, Jiali Cheng, et al.
Canadian Journal of Physiology and Pharmacology|December 8, 2016
Mitochondrial ROS, uncoupled from ATP synthesis, determine endothelial activation for both physiological recruitment of patrolling cells and pathological recruitment of inflammatory cellsXinyuan Li, Pu Fang, William Y Yang, et al.
Advances in Experimental Medicine and Biology|May 29, 2017
Mitochondrial Proton Leak Plays a Critical Role in Pathogenesis of Cardiovascular DiseasesJiali Cheng, Gayani Nanayakkara, Ying Shao, et al.
Frontiers in Immunology|February 14, 2018
GATA3, HDAC6, and BCL6 Regulate FOXP3+ Treg Plasticity and Determine Treg Conversion into Either Novel Antigen-Presenting Cell-Like Treg or Th1-TregKeman Xu, William Y Yang, Gayani Kanchana Nanayakkara, et al.
International Journal of Drug Discovery and Pharmacology|February 10, 2025
Immune Checkpoints Are New Therapeutic Targets in Regulating Cardio-, and Cerebro-Vascular Diseases and CD4<sup>+</sup>Foxp3<sup>+</sup> Regulatory T Cell ImmunosuppressionYing Shao, William Y Yang, Gayani Nanayakkara, et al.
Frontiers in Cardiovascular Medicine|December 11, 2024
Perspective: Pathological transdifferentiation-a novel therapeutic target for cardiovascular diseases and chronic inflammationWilliam Y Yang, Mohammed Ben Issa, Fatma Saaoud, et al.
Arteriosclerosis, Thrombosis, and Vascular Biology|May 28, 2020
Vascular Endothelial Cells and Innate ImmunityYing Shao, Jason Saredy, William Y Yang, et al.
Pageof 4

Showing results (1-10 of 34) with videos related to

Sort By:
Pageof 4
Cytokine|June 27, 2017
IL-35, as a newly proposed homeostasis-associated molecular pattern, plays three major functions including anti-inflammatory initiator, effector, and blocker in cardiovascular diseasesXinyuan Li, Pu Fang, William Y Yang, et al.
Burns & Trauma|December 2, 2015
Pathological conditions re-shape physiological Tregs into pathological TregsWilliam Y Yang, Ying Shao, Jahaira Lopez-Pastrana, et al.
Redox Biology|November 16, 2019
Anti-inflammatory cytokines IL-35 and IL-10 block atherogenic lysophosphatidylcholine-induced, mitochondrial ROS-mediated innate immune activation, but spare innate immune memory signature in endothelial cellsXinyuan Li, Pu Fang, Yu Sun, et al.
Antioxidants & Redox Signaling|March 23, 2017
Lysophospholipids and Their Receptors Serve as Conditional DAMPs and DAMP Receptors in Tissue Oxidative and Inflammatory InjuryYing Shao, Gayani Nanayakkara, Jiali Cheng, et al.
Canadian Journal of Physiology and Pharmacology|December 8, 2016
Mitochondrial ROS, uncoupled from ATP synthesis, determine endothelial activation for both physiological recruitment of patrolling cells and pathological recruitment of inflammatory cellsXinyuan Li, Pu Fang, William Y Yang, et al.
Advances in Experimental Medicine and Biology|May 29, 2017
Mitochondrial Proton Leak Plays a Critical Role in Pathogenesis of Cardiovascular DiseasesJiali Cheng, Gayani Nanayakkara, Ying Shao, et al.
Frontiers in Immunology|February 14, 2018
GATA3, HDAC6, and BCL6 Regulate FOXP3+ Treg Plasticity and Determine Treg Conversion into Either Novel Antigen-Presenting Cell-Like Treg or Th1-TregKeman Xu, William Y Yang, Gayani Kanchana Nanayakkara, et al.
International Journal of Drug Discovery and Pharmacology|February 10, 2025
Immune Checkpoints Are New Therapeutic Targets in Regulating Cardio-, and Cerebro-Vascular Diseases and CD4<sup>+</sup>Foxp3<sup>+</sup> Regulatory T Cell ImmunosuppressionYing Shao, William Y Yang, Gayani Nanayakkara, et al.
Frontiers in Cardiovascular Medicine|December 11, 2024
Perspective: Pathological transdifferentiation-a novel therapeutic target for cardiovascular diseases and chronic inflammationWilliam Y Yang, Mohammed Ben Issa, Fatma Saaoud, et al.
Arteriosclerosis, Thrombosis, and Vascular Biology|May 28, 2020
Vascular Endothelial Cells and Innate ImmunityYing Shao, Jason Saredy, William Y Yang, et al.
Pageof 4