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Nature Communications|May 24, 2023
Phospholipase D3 degrades mitochondrial DNA to regulate nucleotide signaling and APP metabolismZoë P Van Acker, Anika Perdok, Ruben Hellemans, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|June 5, 2012
Peptides based on the presenilin-APP binding domain inhibit APP processing and Aβ production through interfering with the APP transmembrane domainCary Esselens, Ragna Sannerud, Rodrigo Gallardo, et al.
Proceedings of the National Academy of Sciences of the United States of America|January 25, 2005
Differential contribution of the three Aph1 genes to gamma-secretase activity in vivoLutgarde Serneels, Tim Dejaegere, Katleen Craessaerts, et al.
Molecular Biology of the Cell|April 24, 2015
Primary cilium suppression by SREBP1c involves distortion of vesicular trafficking by PLA2G3Hannah Laura Gijs, Nicolas Willemarck, Frank Vanderhoydonc, et al.
Journal of Cell Science|February 3, 2019
The PIKfyve complex regulates the early melanosome homeostasis required for physiological amyloid formationChristin Bissig, Pauline Croisé, Xavier Heiligenstein, et al.
The Journal of Biological Chemistry|February 14, 2009
ADAM10, the rate-limiting protease of regulated intramembrane proteolysis of Notch and other proteins, is processed by ADAMS-9, ADAMS-15, and the gamma-secretaseThomas Tousseyn, Amantha Thathiah, Ellen Jorissen, et al.
Elife|July 8, 2020
Super-resolution microscopy reveals majorly mono- and dimeric presenilin1/γ-secretase at the cell surfaceAbril Angélica Escamilla-Ayala, Ragna Sannerud, Magali Mondin, et al.
Biological Chemistry|May 16, 2016
Tetraspanin 8 is an interactor of the metalloprotease meprin β within tetraspanin-enriched microdomainsFrederike Schmidt, Miryam Müller, Johannes Prox, et al.
Biological Chemistry|May 16, 2016
Tetraspanin 8 is an interactor of the metalloprotease meprin β within tetraspanin-enriched microdomainsFrederike Schmidt, Miryam Müller, Johannes Prox, et al.
The Journal of Cell Biology|July 4, 2012
Lysosomal calcium homeostasis defects, not proton pump defects, cause endo-lysosomal dysfunction in PSEN-deficient cellsKatrijn Coen, Ronald S Flannagan, Szilvia Baron, et al.
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