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Cell and Tissue Research|November 7, 2013
TGF-ß induces Lysyl hydroxylase 2b in human synovial osteoarthritic fibroblasts through ALK5 signalingDennis F G Remst, Esmeralda N Blaney Davidson, Elly L Vitters, et al.
Arthritis and Rheumatism|February 16, 2011
Essential role of microRNA-155 in the pathogenesis of autoimmune arthritis in miceStephan Blüml, Michael Bonelli, Birgit Niederreiter, et al.
Arthritis and Rheumatism|July 28, 2010
S100A8 causes a shift toward expression of activatory Fcγ receptors on macrophages via toll-like receptor 4 and regulates Fcγ receptor expression in synovium during chronic experimental arthritisPeter L van Lent, Lilyanne C Grevers, Rik Schelbergen, et al.
Annals of the Rheumatic Diseases|April 9, 2011
A novel Saa3-promoter reporter distinguishes inflammatory subtypes in experimental arthritis and human synovial fibroblastsJeroen Geurts, Eline A Vermeij, Dirk Pohlers, et al.
Arthritis Research & Therapy|February 18, 2010
Functional consequences of DECTIN-1 early stop codon polymorphism Y238X in rheumatoid arthritisTheo S Plantinga, Jaap Fransen, Nozomi Takahashi, et al.
Arthritis and Rheumatism|December 30, 2006
Crucial role of macrophages in matrix metalloproteinase-mediated cartilage destruction during experimental osteoarthritis: involvement of matrix metalloproteinase 3Arjen B Blom, Peter L van Lent, Sten Libregts, et al.
Rheumatology (Oxford, England)|December 16, 2015
Wnts talking with the TGF-β superfamily: WISPers about modulation of osteoarthritisMartijn H van den Bosch, Teresa A Gleissl, Arjen B Blom, et al.
Osteoarthritis and Cartilage|January 11, 2005
Male IL-6 gene knock out mice developed more advanced osteoarthritis upon agingAlfons S K de Hooge, Fons A J van de Loo, Miranda B Bennink, et al.
Plos One|October 27, 2010
Role of interleukin 17 in arthritis chronicity through survival of synoviocytes via regulation of synoviolin expressionMyew-Ling Toh, Gaelle Gonzales, Marije I Koenders, et al.
The American Journal of Pathology|October 17, 2009
Local interleukin-1-driven joint pathology is dependent on toll-like receptor 4 activationShahla Abdollahi-Roodsaz, Leo A B Joosten, Marije I Koenders, et al.
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