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Chemical Research in Toxicology|July 9, 2015
The Role of CD8 T Cells in Amodiaquine-Induced Liver Injury in PD1-/- Mice Cotreated with Anti-CTLA-4Alastair Mak, Jack UetrechtJournal of Immunotoxicology|November 27, 2014
Immunization with amodiaquine-modified hepatic proteins prevents amodiaquine-induced liver injuryAlastair Mak, Jack UetrechtChemical Research in Toxicology|October 25, 2016
How Reactive Metabolites Induce an Immune Response That Sometimes Leads to an Idiosyncratic Drug ReactionTiffany Cho, Jack UetrechtJournal of Clinical and Translational Research|March 16, 2019
Immune mechanisms of idiosyncratic drug-induced liver injuryAlastair Mak, Jack UetrechtChemical Research in Toxicology|May 20, 2017
Supernatant from Hepatocyte Cultures with Drugs That Cause Idiosyncratic Liver Injury Activates Macrophage InflammasomesRyuji Kato, Jack UetrechtJournal of Immunotoxicology|October 9, 2012
A novel T(H)17-type cell is rapidly increased in the liver in response to acetaminophen-induced liver injury: T(H)17 cells and the innate immune responseXu Zhu, Jack UetrechtAnnals of Translational Medicine|May 14, 2021
Mechanism of idiosyncratic drug induced liver injury (DILI): unresolved basic issuesRolf Teschke, Jack UetrechtExpert Opinion on Drug Metabolism & Toxicology|November 27, 2014
Current understanding of the mechanisms of idiosyncratic drug-induced agranulocytosisAlexander Johnston, Jack UetrechtChemical Research in Toxicology|November 4, 2015
The Combination of Anti-CTLA-4 and PD1-/- Mice Unmasks the Potential of Isoniazid and Nevirapine To Cause Liver InjuryAlastair Mak, Jack UetrechtJournal of Immunotoxicology|January 22, 2019
Involvement of CCL2/CCR2 macrophage recruitment in amodiaquine-induced liver injuryAlastair Mak, Jack UetrechtPageof 10