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The Journal of Biological Chemistry|March 19, 2003
Poly(ADP-ribose) polymerase-1 is a positive regulator of the p53-mediated G1 arrest response following ionizing radiationSonya Wieler, Jean-Philippe Gagné, Homayoun Vaziri, et al.Molecular and Cellular Biology|August 19, 2005
p53-Dependent transcriptional repression of c-myc is required for G1 cell cycle arrestJenny S L Ho, Weili Ma, Daniel Y L Mao, et al.Cell Death & Disease|January 10, 2019
Erythropoietin inhibits chemotherapy-induced cell death and promotes a senescence-like state in leukemia cellsThuc-Nghi Duc Pham, Weili Ma, David Miller, et al.Experimental Cell Research|April 2, 2002
Expression of different mutant p53 transgenes in neuroblastoma cells leads to different cellular responses to genotoxic agentsSuman Gangopadhyay, Farid Jalali, Danny Reda, et al.Oncogene|May 29, 2003
Resistance to DNA-damaging agents is discordant from experimental metastatic capacity in MEF ras-transformants-expressing gain of function MTp53Robert G Bristow, James Peacock, Anne Jang, et al.Molecular and Cellular Biology|May 10, 2017
Progerin-Induced Replication Stress Facilitates Premature Senescence in Hutchinson-Gilford Progeria SyndromeKeith Wheaton, Denise Campuzano, Weili Ma, et al.Cell Cycle (Georgetown, Tex.)|April 23, 2013
Cdk9 phosphorylates Pirh2 protein and prevents degradation of p53 proteinAsen Bagashev, Shongshan Fan, Ruma Mukerjee, et al.Proceedings of the National Academy of Sciences of the United States of America|September 27, 2005
Apoptosis caused by p53-induced protein with death domain (PIDD) depends on the death adapter protein RAIDDChristina Berube, Louis-Martin Boucher, Weili Ma, et al.Cell|March 26, 2003
Pirh2, a p53-induced ubiquitin-protein ligase, promotes p53 degradationRoger P Leng, Yunping Lin, Weili Ma, et al.Cell Death & Disease|August 5, 2023
Heterogeneity in leukemia cells that escape drug-induced senescence-like stateDavid Miller, Kyra Kerkhofs, Farnoosh Abbas-Aghababazadeh, et al.Pageof 3