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Wolfram H Zimmermann

Showing results (1-10 of 38) with videos related to

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Circulation Research|December 13, 2005
Engineering myocardial tissueThomas Eschenhagen, Wolfram H Zimmermann
Circulation Research|March 18, 2006
Electrical coupling of cardiac myocyte cell sheets to the heartThomas Eschenhagen, Wolfram H Zimmermann, André G Kléber
The Journal of Biological Chemistry|January 11, 2005
Systematic evaluation of anti-apoptotic growth factor signaling in vascular smooth muscle cells. Only phosphatidylinositol 3'-kinase is importantMarius Vantler, Evren Caglayan, Wolfram H Zimmermann, et al.
Basic Research in Cardiology|September 2, 2017
Cardiac optogenetics: using light to monitor cardiac physiologyCharlotte D Koopman, Wolfram H Zimmermann, Thomas Knöpfel, et al.
Cardiovascular Research|December 27, 2005
Alterations of the preproenkephalin system in cardiac hypertrophy and its role in atrioventricular conductionJoachim Weil, Oliver Zolk, Jens Griepentrog, et al.
Circulation Research|December 22, 2007
Thyroid hormone regulates developmental titin isoform transitions via the phosphatidylinositol-3-kinase/ AKT pathwayMartina Krüger, Christine Sachse, Wolfram H Zimmermann, et al.
Cellular Physiology and Biochemistry : International Journal of Experimental Cellular Physiology, Biochemistry, and Pharmacology|August 7, 2014
Lights on for HIF-1α: genetically enhanced mouse cardiomyocytes for heart tissue imagingAmke R Hesse, Elif Levent, Anke Zieseniss, et al.
Stem Cell Research|August 11, 2024
Human induced pluripotent stem cells for live cell cycle monitoring and endogenous gene activationRosa Kim, Sebastian H Nagel, Norman Y Liaw, et al.
Journal of Molecular and Cellular Cardiology|March 24, 2010
PDGF-BB protects cardiomyocytes from apoptosis and improves contractile function of engineered heart tissueMarius Vantler, Bijoy Chandapillai Karikkineth, Hiroshi Naito, et al.
Basic Research in Cardiology|December 22, 2010
Diastolic dysfunction and arrhythmias caused by overexpression of CaMKIIδ(C) can be reversed by inhibition of late Na(+) currentSamuel Sossalla, Ulrike Maurer, Hanna Schotola, et al.
Pageof 4

Showing results (1-10 of 38) with videos related to

Sort By:
Pageof 4
Circulation Research|December 13, 2005
Engineering myocardial tissueThomas Eschenhagen, Wolfram H Zimmermann
Circulation Research|March 18, 2006
Electrical coupling of cardiac myocyte cell sheets to the heartThomas Eschenhagen, Wolfram H Zimmermann, André G Kléber
The Journal of Biological Chemistry|January 11, 2005
Systematic evaluation of anti-apoptotic growth factor signaling in vascular smooth muscle cells. Only phosphatidylinositol 3'-kinase is importantMarius Vantler, Evren Caglayan, Wolfram H Zimmermann, et al.
Basic Research in Cardiology|September 2, 2017
Cardiac optogenetics: using light to monitor cardiac physiologyCharlotte D Koopman, Wolfram H Zimmermann, Thomas Knöpfel, et al.
Cardiovascular Research|December 27, 2005
Alterations of the preproenkephalin system in cardiac hypertrophy and its role in atrioventricular conductionJoachim Weil, Oliver Zolk, Jens Griepentrog, et al.
Circulation Research|December 22, 2007
Thyroid hormone regulates developmental titin isoform transitions via the phosphatidylinositol-3-kinase/ AKT pathwayMartina Krüger, Christine Sachse, Wolfram H Zimmermann, et al.
Cellular Physiology and Biochemistry : International Journal of Experimental Cellular Physiology, Biochemistry, and Pharmacology|August 7, 2014
Lights on for HIF-1α: genetically enhanced mouse cardiomyocytes for heart tissue imagingAmke R Hesse, Elif Levent, Anke Zieseniss, et al.
Stem Cell Research|August 11, 2024
Human induced pluripotent stem cells for live cell cycle monitoring and endogenous gene activationRosa Kim, Sebastian H Nagel, Norman Y Liaw, et al.
Journal of Molecular and Cellular Cardiology|March 24, 2010
PDGF-BB protects cardiomyocytes from apoptosis and improves contractile function of engineered heart tissueMarius Vantler, Bijoy Chandapillai Karikkineth, Hiroshi Naito, et al.
Basic Research in Cardiology|December 22, 2010
Diastolic dysfunction and arrhythmias caused by overexpression of CaMKIIδ(C) can be reversed by inhibition of late Na(+) currentSamuel Sossalla, Ulrike Maurer, Hanna Schotola, et al.
Pageof 4