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Journal of Neurochemistry|March 25, 2016
Genetics of FTLD: overview and what else we can expect from genetic studiesCyril Pottier, Thomas A Ravenscroft, Monica Sanchez-Contreras, et al.Antimicrobial Agents and Chemotherapy|August 15, 2022
Genomic Analysis of a Strain Collection Containing Multidrug-, Extensively Drug-, Pandrug-, and Carbapenem-Resistant Modern Clinical Isolates of Acinetobacter baumanniiAdam Valcek, Kristina Nesporova, Clémence Whiteway, et al.Molecular Neurodegeneration|April 24, 2025
Increased TMEM106B levels lead to lysosomal dysfunction which affects synaptic signaling and neuronal healthJolien Perneel, Miranda Lastra Osua, Sara Alidadiani, et al.Frontiers in Neurology|April 20, 2023
Case report: TMEM106B haplotype alters penetrance of GRN mutation in frontotemporal dementia familyJolien Perneel, Masood Manoochehri, Edward D Huey, et al.Biorxiv : the Preprint Server for Biology|February 6, 2026
Distinct TAF15 amyloid filament folds define multiple subtypes of FTLD-TAF15Stephan Tetter, Nikhil R Varghese, Alexey G Murzin, et al.Neuro-Degenerative Diseases|February 11, 2010
Traumatic brain injury may increase the risk for frontotemporal dementia through reduced progranulinAli Jawaid, Rosa Rademakers, Joseph S Kass, et al.Journal of Neuropathology and Experimental Neurology|February 11, 2015
Pathologic staging of white matter lesions in adult-onset leukoencephalopathy/leukodystrophy with axonal spheroidsMurad Alturkustani, Julia Keith, Lili-Naz Hazrati, et al.Genome Research|June 13, 2019
Structural variants identified by Oxford Nanopore PromethION sequencing of the human genomeWouter De Coster, Peter De Rijk, Arne De Roeck, et al.Molecular Neurodegeneration|June 23, 2018
Partial Tmem106b reduction does not correct abnormalities due to progranulin haploinsufficiencyAndrew E Arrant, Alexandra M Nicholson, Xiaolai Zhou, et al.Nature Reviews. Neurology|April 14, 2011
Corticobasal degeneration: a pathologically distinct 4R tauopathyNaomi Kouri, Jennifer L Whitwell, Keith A Josephs, et al.Pageof 41