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Journal of the National Cancer Institute|November 12, 1998
Macrophage role in the anti-prostate cancer response to one class of antiangiogenic agentsI B Joseph, J T IsaacsCancer Research|February 1, 1984
Biochemical methods for predicting metastatic ability of prostatic cancer utilizing the dunning R-3327 rat prostatic adenocarcinoma system as a modelF C Lowe, J T IsaacsEndocrinology|February 1, 1984
Comparative aspects of prostatic growth and androgen metabolism with aging in the dog versus the ratS J Berry, J T IsaacsCancer Research|December 1, 1981
Adaptation versus selection as the mechanism responsible for the relapse of prostatic cancer to androgen ablation therapy as studied in the Dunning R-3327-H adenocarcinomaJ T Isaacs, D S CoffeyThe Journal of Urology|May 1, 1988
Relationship between tumor size and the curability of metastatic prostatic cancer by surgery alone or in combination with adjuvant chemotherapyJ M Henry, J T IsaacsThe Prostate|September 1, 1996
The antiangiogenic agent linomide inhibits tumor necrosis factor-alpha secretion via inhibition of its synthesisI B Joseph, J T IsaacsEndocrine-Related Cancer|March 27, 2002
Mechanisms involved in the progression of androgen-independent prostate cancers: it is not only the cancer cell's faultJ T Arnold, J T IsaacsThe Prostate|January 1, 1989
Prostatic growth effects of rat urogenital sinus and human prostatic tissue in the ratJ P Jarow, J T IsaacsCancer Research|December 1, 1985
Effectiveness of complete versus partial androgen withdrawal therapy for the treatment of prostatic cancer as studied in the Dunning R-3327 system of rat prostatic adenocarcinomasW J Ellis, J T IsaacsCancer Research|October 1, 1990
Genetic instability and the acquisition of metastatic ability by rat mammary cancer cells following v-H-ras oncogene transfectionT Ichikawa, N Kyprianou, J T IsaacsPageof 170