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The Journal of Cell Biology|October 8, 1998
Bax-induced cytochrome C release from mitochondria is independent of the permeability transition pore but highly dependent on Mg2+ ionsR Eskes, B Antonsson, A Osen-Sand, et al.The Journal of Cell Biology|March 23, 1999
Bid-induced conformational change of Bax is responsible for mitochondrial cytochrome c release during apoptosisS Desagher, A Osen-Sand, A Nichols, et al.Journal of Bioenergetics and Biomembranes|June 1, 1995
Investigations of the inhibitory effect of propranolol, chlorpromazine, quinine, and dicyclohexylcarbodiimide on the swelling of yeast mitochondria in potassium acetate. Evidences for indirect effects mediated by the lipid phaseX Roucou, S Manon, M GuérinFEBS Letters|May 8, 1995
ATP opens an electrophoretic potassium transport pathway in respiring yeast mitochondriaX Roucou, S Manon, M GuerinBiochemistry and Molecular Biology International|October 7, 1997
Modulation of the electrophoretic ATP-induced K(+)-transport in yeast mitochondria by delta pHX Roucou, S Manon, M GuérinBiochimica Et Biophysica Acta|February 21, 1997
Conditions allowing different states of ATP- and GDP-induced permeability in mitochondria from different strains of Saccharomyces cerevisiaeX Roucou, S Manon, M GuérinCell and Tissue Research|December 6, 2001
Bax and other pro-apoptotic Bcl-2 family "killer-proteins" and their victim the mitochondrionB AntonssonBiochimica Et Biophysica Acta|November 25, 1997
Phosphatidylinositol synthase from mammalian tissuesB AntonssonThe Journal of Biological Chemistry|October 6, 1997
Bcl-2 undergoes phosphorylation by c-Jun N-terminal kinase/stress-activated protein kinases in the presence of the constitutively active GTP-binding protein Rac1K Maundrell, B Antonsson, E Magnenat, et al.Trends in Cell Biology|August 10, 2000
Mitochondria as the central control point of apoptosisS Desagher, J C MartinouPageof 13