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Cell Death and Differentiation|February 4, 2017
Autophagy induced by DAMPs facilitates the inflammation response in lungs undergoing ischemia-reperfusion injury through promoting TRAF6 ubiquitinationXingguang Liu, Hao Cao, Jian Li, et al.
Journal of Medical Virology|September 20, 2022
Single-cell transcriptome atlas reveals protective characteristics of COVID-19 mRNA vaccineYong Tan, Shuaiyao Lu, Bo Wang, et al.
The Journal of Experimental Medicine|March 8, 2019
KAT8 selectively inhibits antiviral immunity by acetylating IRF3Wanwan Huai, Xingguang Liu, Chunmei Wang, et al.
Biochemical Pharmacology|May 20, 2023
IRG1 prevents excessive inflammatory responses and cardiac dysfunction after myocardial injuryXuewen Duan, Meiling Hu, Linshan Yang, et al.
Journal of Immunology (Baltimore, Md. : 1950)|September 24, 2011
CMRF-35-like molecule 3 preferentially promotes TLR9-triggered proinflammatory cytokine production in macrophages by enhancing TNF receptor-associated factor 6 ubiquitinationYanan Wu, Xuhui Zhu, Nan Li, et al.
Nature Immunology|July 26, 2011
The microRNA miR-29 controls innate and adaptive immune responses to intracellular bacterial infection by targeting interferon-γFeng Ma, Sheng Xu, Xingguang Liu, et al.
Cell Research|June 20, 2019
Interferon-inducible cytoplasmic lncLrrc55-AS promotes antiviral innate responses by strengthening IRF3 phosphorylationYumei Zhou, Mengxuan Li, Yiquan Xue, et al.
Experimental & Molecular Medicine|December 9, 2022
Loss of KDM5B ameliorates pathological cardiac fibrosis and dysfunction by epigenetically enhancing ATF3 expressionBo Wang, Yong Tan, Yunkai Zhang, et al.
Cellular & Molecular Immunology|June 15, 2019
KDM2B promotes IL-6 production and inflammatory responses through Brg1-mediated chromatin remodelingQingqing Zhou, Yunkai Zhang, Bo Wang, et al.
The Journal of Biological Chemistry|April 24, 2013
Immune responsive gene 1 (IRG1) promotes endotoxin tolerance by increasing A20 expression in macrophages through reactive oxygen speciesYingke Li, Peng Zhang, Chengcai Wang, et al.
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