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Current Drug Targets|June 2, 2015
HIV-1 Tat-Mediated Calcium Dysregulation and Neuronal Dysfunction in Vulnerable Brain RegionsXiu-Ti HuAlzheimers Disease & Dementia|September 24, 2020
A Novel Concept is Needed for Combating Alzheimer's Disease and NeuroHIVXiu-Ti HuMolecular Neurobiology|May 24, 2007
Cocaine withdrawal and neuro-adaptations in ion channel functionXiu-Ti HuTrends in Neurosciences|September 8, 2006
Exciting inhibition in psychostimulant addictionPeter W Kalivas, Xiu-Ti HuNeuropsychopharmacology : Official Publication of the American College of Neuropsychopharmacology|February 24, 2005
Repeated cocaine administration decreases calcineurin (PP2B) but enhances DARPP-32 modulation of sodium currents in rat nucleus accumbens neuronsXiu-Ti Hu, Kerstin Ford, Francis J WhiteJournal of Neurophysiology|August 28, 2004
Repeated cocaine administration suppresses HVA-Ca2+ potentials and enhances activity of K+ channels in rat nucleus accumbens neuronsXiu-Ti Hu, Somnath Basu, Francis J WhiteFrontiers in Pharmacology|February 15, 2021
Triumeq Increases Excitability of Pyramidal Neurons in the Medial Prefrontal Cortex by Facilitating Voltage-Gated Ca2+ Channel FunctionLihua Chen, Lena Al-Harthi, Xiu-Ti HuThe Journal of Neuroscience : the Official Journal of the Society for Neuroscience|April 9, 2005
Repeated cocaine administration increases voltage-sensitive calcium currents in response to membrane depolarization in medial prefrontal cortex pyramidal neuronsFernando J Nasif, Xiu-Ti Hu, Francis J WhiteJournal of Neurophysiology|August 4, 2006
Dopamine D(2) receptor modulation of K(+) channel activity regulates excitability of nucleus accumbens neurons at different membrane potentialsMariela F Perez, Francis J White, Xiu-Ti HuSynapse (New York, N.Y.)|April 11, 2009
Plasticity of L-type Ca2+ channels after cocaine withdrawalKerstin A Ford, Marina E Wolf, Xiu-Ti HuPageof 4