Y J De Kok - JoVE Visualize

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Human Mutation|January 1, 1997

The molecular basis of X-linked deafness type 3 (DFN3) in two sporadic cases: identification of a somatic mosaicism for a POU3F4 missense mutationY J de Kok, C W Cremers, H H Ropers, et al.

Cell and Tissue Research|May 1, 1995

Tissue- and cell-specific distribution of creatine kinase B: a new and highly specific monoclonal antibody for use in immunohistochemistryE A Sistermans, Y J de Kok, W Peters, et al.

Molecular and Cellular Biochemistry|February 9, 1995

Production of native creatine kinase B in insect cells using a baculovirus expression vectorY J de Kok, M P Geurds, E A Sistermans, et al.

Pharmacology, Biochemistry, and Behavior|December 1, 1987

A genotype-dependent hippocampal dynorphinergic mechanism controls mouse explorationJ H Van Daal, Y J De Kok, B G Jenks, et al.

Human Molecular Genetics|December 6, 2001

CRB1 has a cytoplasmic domain that is functionally conserved between human and DrosophilaA I den Hollander, K Johnson, Y J de Kok, et al.

The Laryngoscope|September 28, 1999

Progressive cochleovestibular impairment caused by a point mutation in the COCH gene at DFNA9S J Bom, M H Kemperman, Y J De Kok, et al.

Human Molecular Genetics|November 1, 1995

A duplication/paracentric inversion associated with familial X-linked deafness (DFN3) suggests the presence of a regulatory element more than 400 kb upstream of the POU3F4 geneY J de Kok, G F Merkx, S M van der Maarel, et al.

Science (New York, N.Y.)|February 3, 1995

Association between X-linked mixed deafness and mutations in the POU domain gene POU3F4Y J de Kok, S M van der Maarel, M Bitner-Glindzicz, et al.

Genomics|June 22, 1999

Isolation and mapping of novel candidate genes for retinal disorders using suppression subtractive hybridizationA I den Hollander, M A van Driel, Y J de Kok, et al.

Human Molecular Genetics|July 1, 1994

X-linked mixed deafness (DFN3): cloning and characterization of the critical region allows the identification of novel microdeletionsI Huber, M Bitner-Glindzicz, Y J de Kok, et al.

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Showing results (1-10 of 15) with videos related to

Sort By:

Pageof 2

Human Mutation|January 1, 1997

The molecular basis of X-linked deafness type 3 (DFN3) in two sporadic cases: identification of a somatic mosaicism for a POU3F4 missense mutationY J de Kok, C W Cremers, H H Ropers, et al.

Cell and Tissue Research|May 1, 1995

Tissue- and cell-specific distribution of creatine kinase B: a new and highly specific monoclonal antibody for use in immunohistochemistryE A Sistermans, Y J de Kok, W Peters, et al.

Molecular and Cellular Biochemistry|February 9, 1995

Production of native creatine kinase B in insect cells using a baculovirus expression vectorY J de Kok, M P Geurds, E A Sistermans, et al.

Pharmacology, Biochemistry, and Behavior|December 1, 1987

A genotype-dependent hippocampal dynorphinergic mechanism controls mouse explorationJ H Van Daal, Y J De Kok, B G Jenks, et al.

Human Molecular Genetics|December 6, 2001

CRB1 has a cytoplasmic domain that is functionally conserved between human and DrosophilaA I den Hollander, K Johnson, Y J de Kok, et al.

The Laryngoscope|September 28, 1999

Progressive cochleovestibular impairment caused by a point mutation in the COCH gene at DFNA9S J Bom, M H Kemperman, Y J De Kok, et al.

Human Molecular Genetics|November 1, 1995

A duplication/paracentric inversion associated with familial X-linked deafness (DFN3) suggests the presence of a regulatory element more than 400 kb upstream of the POU3F4 geneY J de Kok, G F Merkx, S M van der Maarel, et al.

Science (New York, N.Y.)|February 3, 1995

Association between X-linked mixed deafness and mutations in the POU domain gene POU3F4Y J de Kok, S M van der Maarel, M Bitner-Glindzicz, et al.

Genomics|June 22, 1999

Isolation and mapping of novel candidate genes for retinal disorders using suppression subtractive hybridizationA I den Hollander, M A van Driel, Y J de Kok, et al.

Human Molecular Genetics|July 1, 1994

X-linked mixed deafness (DFN3): cloning and characterization of the critical region allows the identification of novel microdeletionsI Huber, M Bitner-Glindzicz, Y J de Kok, et al.

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