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Physical Review Letters|April 9, 2021
Study of the K_{L}→π^{0}νν[over ¯] Decay at the J-PARC KOTO ExperimentJ K Ahn, B Beckford, M Campbell, et al.Nature Communications|April 29, 2023
Multi-population genome-wide association study implicates immune and non-immune factors in pediatric steroid-sensitive nephrotic syndromeAlexandra Barry, Michelle T McNulty, Xiaoyuan Jia, et al.Physical Review Letters|February 6, 2019
Search for K_{L}→π^{0}νν[over ¯] and K_{L}→π^{0}X^{0} Decays at the J-PARC KOTO ExperimentJ K Ahn, B Beckford, J Beechert, et al.American Journal of Human Genetics|January 28, 2021
De novo TRIM8 variants impair its protein localization to nuclear bodies and cause developmental delay, epilepsy, and focal segmental glomerulosclerosisPatricia L Weng, Amar J Majmundar, Kamal Khan, et al.Medrxiv : the Preprint Server for Health Sciences|August 14, 2023
Strong protective effect of the APOL1 p.N264K variant against G2-associated focal segmental glomerulosclerosis and kidney diseaseYask Gupta, David J Friedman, Michelle McNulty, et al.Journal of the American Society of Nephrology : JASN|February 18, 2021
Copy Number Variant Analysis and Genome-wide Association Study Identify Loci with Large Effect for Vesicoureteral RefluxMiguel Verbitsky, Priya Krithivasan, Ekaterina Batourina, et al.Journal of the American Society of Nephrology : JASN|March 30, 2023
Rare Single Nucleotide and Copy Number Variants and the Etiology of Congenital Obstructive Uropathy: Implications for Genetic DiagnosisDina F Ahram, Tze Y Lim, Juntao Ke, et al.Nature Communications|November 30, 2023
Strong protective effect of the APOL1 p.N264K variant against G2-associated focal segmental glomerulosclerosis and kidney diseaseYask Gupta, David J Friedman, Michelle T McNulty, et al.Updates in Surgery|March 6, 2026
MYELOstudy: lymph node stage of aggressiveness in medullary thyroid cancer-a retrospective multi-center study analysisTheodossis Papavramidis, Angeliki Chorti, Sohail Bakkar, et al.The Journal of Clinical Investigation|March 28, 2023
CERT1 mutations perturb human development by disrupting sphingolipid homeostasisCharlotte Gehin, Museer A Lone, Winston Lee, et al.Pageof 125