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Proceedings of the National Academy of Sciences of the United States of America|January 19, 2000
Proapoptotic BH3-only Bcl-2 family members induce cytochrome c release, but not mitochondrial membrane potential loss, and do not directly modulate voltage-dependent anion channel activityS Shimizu, Y TsujimotoCell Death and Differentiation|February 15, 2001
VDAC regulation by the Bcl-2 family of proteinsY Tsujimoto, S ShimizuCell Death and Differentiation|October 26, 2005
Another way to die: autophagic programmed cell deathY Tsujimoto, S ShimizuRinsho Byori. the Japanese Journal of Clinical Pathology|May 1, 1997
[Molecular biology of apoptosis]Y Eguchi, S Shimizu, Y TsujimotoOncogene|September 12, 2000
Bax and Bcl-xL independently regulate apoptotic changes of yeast mitochondria that require VDAC but not adenine nucleotide translocatorS Shimizu, Y Shinohara, Y TsujimotoCancer Research|May 15, 1997
Intracellular ATP levels determine cell death fate by apoptosis or necrosisY Eguchi, S Shimizu, Y TsujimotoNature|June 12, 1999
Bcl-2 family proteins regulate the release of apoptogenic cytochrome c by the mitochondrial channel VDACS Shimizu, M Narita, Y TsujimotoThe Journal of Biological Chemistry|April 15, 2000
Electrophysiological study of a novel large pore formed by Bax and the voltage-dependent anion channel that is permeable to cytochrome cS Shimizu, T Ide, T Yanagida, et al.Proceedings of the National Academy of Sciences of the United States of America|March 29, 2000
BH4 domain of antiapoptotic Bcl-2 family members closes voltage-dependent anion channel and inhibits apoptotic mitochondrial changes and cell deathS Shimizu, A Konishi, T Kodama, et al.Pageof 153