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Bioorganic & Medicinal Chemistry Letters|November 13, 2003
Neuroprotective or neurotoxic activity of 1-methyl-1,2,3,4-tetrahydroisoquinoline and related compoundsKatsuhiro Okuda, Yaichiro Kotake, Shigeru Ohta
Metallomics : Integrated Biometal Science|March 29, 2013
AMP-activated protein kinase-mediated glucose transport as a novel target of tributyltin in human embryonic carcinoma cellsShigeru Yamada, Yaichiro Kotake, Yuko Sekino, et al.
Toxicology and Applied Pharmacology|July 14, 2009
Long-term exposure to endogenous levels of tributyltin decreases GluR2 expression and increases neuronal vulnerability to glutamateYusuke Nakatsu, Yaichiro Kotake, Tomoko Takishita, et al.
Toxicology and Applied Pharmacology|May 31, 2008
Activation of AMP-activated protein kinase by tributyltin induces neuronal cell deathYusuke Nakatsu, Yaichiro Kotake, Atsuko Hino, et al.
Neurotoxicology|July 25, 2006
Cytotoxicity of 17 tetrahydroisoquinoline derivatives in SH-SY5Y human neuroblastoma cells is related to mitochondrial NADH-ubiquinone oxidoreductase inhibitionYaichiro Kotake, Yoko Sekiya, Katsuhiro Okuda, et al.
The Journal of Toxicological Sciences|September 23, 2014
Detection of a novel neurotoxic metabolite of Parkinson's disease-related neurotoxin, 1-benzyl-1,2,3,4- tetrahydroisoquinolineYaichiro Kotake, Yoko Sekiya, Katsuhiro Okuda, et al.
Biological & Pharmaceutical Bulletin|March 3, 2017
Lead-Induced ERK Activation Is Mediated by GluR2 Non-containing AMPA Receptor in Cortical NeuronsKeishi Ishida, Yaichiro Kotake, Seigo Sanoh, et al.
The Journal of Toxicological Sciences|April 8, 2010
Involvement of autophagy via mammalian target of rapamycin (mTOR) inhibition in tributyltin-induced neuronal cell deathYusuke Nakatsu, Yaichiro Kotake, Naoko Takai, et al.
The Journal of Toxicological Sciences|March 1, 2023
Tributyltin activates the Keap1-Nrf2 pathway via a macroautophagy-independent reduction in Keap1Misaki Hatano, Shunichi Hatamiya, Masatsugu Miyara, et al.
The Journal of Toxicological Sciences|July 1, 2026
Linalool inhalation‒induced anxiety-like effects occur independently of olfactory input and require cytochrome P450 activityHideaki Sato, Ami Oguro, Masatsugu Miyara, et al.
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