Showing results (1-10 of 20) with videos related to

Sort By:
Pageof 2
Journal of Virology|May 2, 2008
Genetic interactions among the West Nile virus methyltransferase, the RNA-dependent RNA polymerase, and the 5' stem-loop of genomic RNABo Zhang, Hongping Dong, Yangsheng Zhou, et al.
International Journal of Biological Macromolecules|May 9, 2024
Self-assembled lignin-based flame retardant hybrids carrying Cu2+ for poly(lactic acid) composites with improved fire safety and mechanical propertiesXianwu Cao, Yangsheng Zhou, Jingshu Huang, et al.
Journal of Virology|February 29, 2008
West Nile virus methyltransferase catalyzes two methylations of the viral RNA cap through a substrate-repositioning mechanismHongping Dong, Suping Ren, Bo Zhang, et al.
British Journal of Haematology|May 8, 2014
Transcriptional repression of plasma cell differentiation is orchestrated by aberrant over-expression of the ETS factor SPIB in Waldenström macroglobulinaemiaYangsheng Zhou, Xia Liu, Lian Xu, et al.
The Journal of General Virology|February 28, 2007
Role of the mutant spectrum in adaptation and replication of West Nile virusAlexander T Ciota, Kiet A Ngo, Amy O Lovelace, et al.
Journal of Virology|August 17, 2006
West Nile virus 5'-cap structure is formed by sequential guanine N-7 and ribose 2'-O methylations by nonstructural protein 5Debashish Ray, Aaloki Shah, Mark Tilgner, et al.
Journal of Virology|February 3, 2007
Structure and function of flavivirus NS5 methyltransferaseYangsheng Zhou, Debashish Ray, Yiwei Zhao, et al.
Virology|December 21, 2006
A mouse cell-adapted NS4B mutation attenuates West Nile virus RNA synthesisFrancesc Puig-Basagoiti, Mark Tilgner, Corey J Bennett, et al.
Clinical Lymphoma, Myeloma & Leukemia|August 23, 2011
Matrix metalloproteinase-8 is overexpressed in Waldenström's macroglobulinemia cells, and specific inhibition of this metalloproteinase blocks release of soluble CD27Yangsheng Zhou, Xia Liu, Lian Xu, et al.
Blood|December 25, 2013
The genomic landscape of Waldenstrom macroglobulinemia is characterized by highly recurring MYD88 and WHIM-like CXCR4 mutations, and small somatic deletions associated with B-cell lymphomagenesisZachary R Hunter, Lian Xu, Guang Yang, et al.
Pageof 2