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Nucleic Acids Research|February 18, 2016
Rad18 confers hematopoietic progenitor cell DNA damage tolerance independently of the Fanconi Anemia pathway in vivoYang Yang, Jonathan C Poe, Lisong Yang, et al.
Nature Communications|July 6, 2016
A neomorphic cancer cell-specific role of MAGE-A4 in trans-lesion synthesisYanzhe Gao, Elizabeth Mutter-Rottmayer, Alicia M Greenwalt, et al.
Genes & Cancer|March 16, 2023
VCP/p97, a pleiotropic protein regulator of the DNA damage response and proteostasis, is a potential therapeutic target in <i>KRAS</i>-mutant pancreatic cancerYe S Lee, Jennifer E Klomp, Clint A Stalnecker, et al.
Protein Science : a Publication of the Protein Society|March 18, 2025
A synthetic heavy chain variable domain antibody library (VHL) provides highly functional antibodies with favorable developabilityGuiying Pang, Ruixue Wang, Hongxu Yang, et al.
JCI Insight|March 28, 2020
Irreversible JNK1-JUN inhibition by JNK-IN-8 sensitizes pancreatic cancer to 5-FU/FOLFOX chemotherapyMatthew B Lipner, Xianlu L Peng, Chong Jin, et al.
Nucleic Acids Research|October 24, 2024
PCNA-binding activity separates RNF168 functions in DNA replication and DNA double-stranded break signalingYang Yang, Deepika Jayaprakash, Satpal S Jhujh, et al.
Research Square|October 27, 2023
Trans-Lesion Synthesis and Mismatch Repair Pathway Crosstalk Defines Chemoresistance and Hypermutation Mechanisms in GlioblastomaXing Cheng, Jing An, Jitong Lou, et al.
Biorxiv : the Preprint Server for Biology|October 31, 2023
Trans-Lesion Synthesis and Mismatch Repair Pathway Crosstalk Defines Chemoresistance and Hypermutation Mechanisms in GlioblastomaXing Cheng, Jing An, Jitong Lou, et al.
Nature Communications|March 4, 2024
Trans-lesion synthesis and mismatch repair pathway crosstalk defines chemoresistance and hypermutation mechanisms in glioblastomaXing Cheng, Jing An, Jitong Lou, et al.
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