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Plos One|March 21, 2015
Interleukin-19 acts as a negative autocrine regulator of activated microgliaHiroshi Horiuchi, Bijay Parajuli, Yue Wang, et al.
Neurochemical Research|June 28, 2018
Lauric Acid Alleviates Neuroinflammatory Responses by Activated Microglia: Involvement of the GPR40-Dependent PathwayYasunori Nishimura, Mitsuaki Moriyama, Kenji Kawabe, et al.
Proceedings of the National Academy of Sciences of the United States of America|August 30, 2006
Inactivation of JNK1 enhances innate IL-10 production and dampens autoimmune inflammation in the brainElise H Tran, Yasu-Taka Azuma, Manchuan Chen, et al.
International Journal of Molecular Sciences|May 25, 2024
Inhibition of Drp1-Filamin Protein Complex Prevents Hepatic Lipid Droplet Accumulation by Increasing Mitochondria-Lipid Droplet ContactKohei Ariyoshi, Kazuhiro Nishiyama, Yuri Kato, et al.
Nitric Oxide : Biology and Chemistry|January 4, 2016
Active site cysteine-null glyceraldehyde-3-phosphate dehydrogenase (GAPDH) rescues nitric oxide-induced cell deathTakeya Kubo, Hidemitsu Nakajima, Masatoshi Nakatsuji, et al.
International Journal of Molecular Sciences|February 24, 2024
Pharmacological Activation of TRPC6 Channel Prevents Colitis ProgressionKazuhiro Nishiyama, Yuri Kato, Akiyuki Nishimura, et al.
Biochemical and Biophysical Research Communications|October 31, 2009
An aggregate-prone mutant of human glyceraldehyde-3-phosphate dehydrogenase augments oxidative stress-induced cell death in SH-SY5Y cellsHidemitsu Nakajima, Wataru Amano, Ayano Fukuhara, et al.
The Journal of Biological Chemistry|July 7, 2007
The active site cysteine of the proapoptotic protein glyceraldehyde-3-phosphate dehydrogenase is essential in oxidative stress-induced aggregation and cell deathHidemitsu Nakajima, Wataru Amano, Akikazu Fujita, et al.
Journal of Pharmacological Sciences|October 29, 2013
Na⁺/Ca²⁺ exchanger 1/2 double-heterozygote knockout mice display increased nitric oxide component and altered colonic motilityKazuhiro Nishiyama, Yasu-Taka Azuma, Satomi Kita, et al.
Biochemical and Biophysical Research Communications|April 10, 2014
Botulinum neurotoxin A subtype 2 reduces pathological behaviors more effectively than subtype 1 in a rat Parkinson's disease modelMasanori Itakura, Tomoko Kohda, Takeya Kubo, et al.
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