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Haematologica|March 18, 2021
Clinical significance of RAS pathway alterations in pediatric acute myeloid leukemiaTaeko Kaburagi, Genki Yamato, Norio Shiba, et al.
Genes, Chromosomes & Cancer|November 30, 2022
UBTF-internal tandem duplication as a novel poor prognostic factor in pediatric acute myeloid leukemiaTaeko Kaburagi, Norio Shiba, Genki Yamato, et al.
Blood Advances|January 10, 2022
Genome-wide DNA methylation analysis in pediatric acute myeloid leukemiaGenki Yamato, Tomoko Kawai, Norio Shiba, et al.
Genes, Chromosomes & Cancer|July 22, 2014
Gene alterations involving the CRLF2-JAK pathway and recurrent gene deletions in Down syndrome-associated acute lymphoblastic leukemia in JapanIsamu Hanada, Kiminori Terui, Fumika Ikeda, et al.
Genes, Chromosomes & Cancer|April 27, 2023
TP53 and RB1 alterations characterize poor prognostic subgroups in pediatric acute myeloid leukemiaYusuke Hara, Norio Shiba, Kenichi Yoshida, et al.
Communications Biology|October 1, 2020
Comprehensive genetic analysis of pediatric germ cell tumors identifies potential drug targetsYasuo Kubota, Masafumi Seki, Tomoko Kawai, et al.
Genes & Development|March 28, 2008
Fbxw7 acts as a critical fail-safe against premature loss of hematopoietic stem cells and development of T-ALLSahoko Matsuoka, Yuichi Oike, Ichiro Onoyama, et al.
Genes, Chromosomes & Cancer|January 8, 2017
ASXL2 mutations are frequently found in pediatric AML patients with t(8;21)/ RUNX1-RUNX1T1 and associated with a better prognosisGenki Yamato, Norio Shiba, Kenichi Yoshida, et al.
The Journal of Allergy and Clinical Immunology|November 16, 2016
Haploinsufficiency of TNFAIP3 (A20) by germline mutation is involved in autoimmune lymphoproliferative syndromeMasatoshi Takagi, Shohei Ogata, Hiroo Ueno, et al.
British Journal of Haematology|June 13, 2021
Droplet digital polymerase chain reaction assay for the detection of the minor clone of KIT D816V in paediatric acute myeloid leukaemia especially showing RUNX1-RUNX1T1 transcriptsKoji Sasaki, Shinichi Tsujimoto, Mayuko Miyake, et al.
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