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Biochemical and Biophysical Research Communications|June 20, 2014
Simplified method for cell-specific gene expression analysis in Caenorhabditis elegansTakuma Sugi, Yasuko Ohtani
Development, Growth & Differentiation|January 14, 2014
Versatile strategy for isolating transcription activator-like effector nuclease-mediated knockout mutants in Caenorhabditis elegansTakuma Sugi, Tetsushi Sakuma, Yasuko Ohtani, et al.
Proceedings of the National Academy of Sciences of the United States of America|November 19, 2014
High-throughput optical quantification of mechanosensory habituation reveals neurons encoding memory in Caenorhabditis elegansTakuma Sugi, Yasuko Ohtani, Yuta Kumiya, et al.
Genes to Cells : Devoted to Molecular & Cellular Mechanisms|June 19, 2007
RBP2 is an MRG15 complex component and down-regulates intragenic histone H3 lysine 4 methylationTomohiro Hayakawa, Yasuko Ohtani, Noriyo Hayakawa, et al.
Molecular and Cellular Biology|September 24, 2008
Balance between distinct HP1 family proteins controls heterochromatin assembly in fission yeastMahito Sadaie, Rika Kawaguchi, Yasuko Ohtani, et al.
Journal of Cell Science|March 25, 2010
MRG15 binds directly to PALB2 and stimulates homology-directed repair of chromosomal breaksTomohiro Hayakawa, Fan Zhang, Noriyo Hayakawa, et al.
The Journal of Biological Chemistry|September 6, 2014
Physical and functional interactions between the histone H3K4 demethylase KDM5A and the nucleosome remodeling and deacetylase (NuRD) complexGohei Nishibuchi, Yukimasa Shibata, Tomohiro Hayakawa, et al.
Molecular Cell|June 26, 2012
Intrinsic nucleic acid-binding activity of Chp1 chromodomain is required for heterochromatic gene silencingMayumi Ishida, Hideaki Shimojo, Aki Hayashi, et al.
Chemphyschem : a European Journal of Chemical Physics and Physical Chemistry|April 30, 2021
Conformational Space Sampled by Domain Reorientation of Linear Diubiquitin Reflected in Its Binding Mode for Target ProteinsXue-Ni Hou, Naotaka Sekiyama, Yasuko Ohtani, et al.
Proceedings of the National Academy of Sciences of the United States of America|September 16, 2022
ALS mutations in the TIA-1 prion-like domain trigger highly condensed pathogenic structuresNaotaka Sekiyama, Kiyofumi Takaba, Saori Maki-Yonekura, et al.
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