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Histology and Histopathology|January 24, 2018
Protective effects of the pericellular matrix of chondrocyte on articular cartilage against the development of osteoarthritisChenlu Liu, Beiyu Wang, Li Xiao, et al.
Cartilage|June 11, 2019
Targeting Discoidin Domain Receptor 2 for the Development of Disease-Modifying Osteoarthritis DrugsLi Xiao, Chenlu Liu, Beiyu Wang, et al.
Arthritis and Rheumatism|June 3, 2010
Attenuation of osteoarthritis progression by reduction of discoidin domain receptor 2 in miceLin Xu, Jacqueline Servais, Ilona Polur, et al.
The American Journal of Pathology|September 11, 2015
Attenuation of the progression of articular cartilage degeneration by inhibition of TGF-β1 signaling in a mouse model of osteoarthritisRebecca Chen, Michelle Mian, Martin Fu, et al.
Human Genetics|December 16, 2003
Localized aggressive periodontitis is linked to human chromosome 1q25Yefu Li, Lin Xu, Hatice Hasturk, et al.
The Journal of Biological Chemistry|October 29, 2004
Activation of the discoidin domain receptor 2 induces expression of matrix metalloproteinase 13 associated with osteoarthritis in miceLin Xu, Haibing Peng, Dongying Wu, et al.
The American Journal of Pathology|August 23, 2011
Intact pericellular matrix of articular cartilage is required for unactivated discoidin domain receptor 2 in the mouse modelLin Xu, Ilona Polur, Jacqueline M Servais, et al.
Arthritis and Rheumatism|October 31, 2007
Increased expression of discoidin domain receptor 2 is linked to the degree of cartilage damage in human knee joints: a potential role in osteoarthritis pathogenesisIlse-Gerlinde Sunk, Klaus Bobacz, Jochen G Hofstaetter, et al.
The American Journal of Pathology|May 4, 2019
High-Temperature Requirement A1 Protease as a Rate-Limiting Factor in the Development of OsteoarthritisPeter H Chen, Tian Tang, Chenlu Liu, et al.
Arthritis and Rheumatism|January 1, 2008
Early-onset degeneration of the intervertebral disc and vertebral end plate in mice deficient in type IX collagenLawrence M Boyd, William J Richardson, Kyle D Allen, et al.
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