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Nature|December 24, 2010
S-glutathionylation uncouples eNOS and regulates its cellular and vascular functionChun-An Chen, Tse-Yao Wang, Saradhadevi Varadharaj, et al.
Nature Communications|July 13, 2026
Genetic or pharmacological inhibition of hepatic TMEM141 attenuates MASH and fibrosis via the ROS-HNF4α signaling pathwayJiayou Wang, Chwen-Lih Chen, Raja Gopoju, et al.
Arteriosclerosis, Thrombosis, and Vascular Biology|June 22, 2013
Mitochondrial oxidative stress corrupts coronary collateral growth by activating adenosine monophosphate activated kinase-α signalingYuh Fen Pung, Wai Johnn Sam, Kelly Stevanov, et al.
Basic Research in Cardiology|February 25, 2016
Differential regulation of TRPV1 channels by H2O2: implications for diabetic microvascular dysfunctionDaniel J DelloStritto, Patrick J Connell, Gregory M Dick, et al.
Basic Research in Cardiology|January 13, 2022
Mechanism of the switch from NO to H<sub>2</sub>O<sub>2</sub> in endothelium-dependent vasodilation in diabetesCody Juguilon, Zhiyuan Wang, Yang Wang, et al.
Basic Research in Cardiology|January 18, 2022
Mitochondrial DNA integrity and function are critical for endothelium-dependent vasodilation in rats with metabolic syndromeTakahiko Kiyooka, Vahagn Ohanyan, Liya Yin, et al.
Basic Research in Cardiology|April 5, 2016
Impaired coronary metabolic dilation in the metabolic syndrome is linked to mitochondrial dysfunction and mitochondrial DNA damageGiacinta Guarini, Takahiko Kiyooka, Vahagn Ohanyan, et al.
European Heart Journal|May 12, 2023
Takotsubo syndrome is a coronary microvascular disease: experimental evidenceFeng Dong, Liya Yin, Hamayak Sisakian, et al.
Nature Immunology|August 12, 2020
IL-1 induces mitochondrial translocation of IRAK2 to suppress oxidative metabolism in adipocytesHao Zhou, Han Wang, Minjia Yu, et al.
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