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Scientific Reports|March 3, 2017
Rhein Inhibits Autophagy in Rat Renal Tubular Cells by Regulation of AMPK/mTOR SignalingYue Tu, Liubao Gu, Diping Chen, et al.
British Journal of Pharmacology|July 24, 2010
Disruption of gap junctions attenuates aminoglycoside-elicited renal tubular cell injuryJian Yao, Tao Huang, Xin Fang, et al.
Frontiers in Pharmacology|February 11, 2026
Rhein protects against renal aging and fibrotic injury by multiple targets through inhibition of TNF-α-mediated autophagy and necroptosis crosstalkYue Tu, Wenjie Liu, Tianqing Sang, et al.
Nephron. Experimental Nephrology|February 22, 2005
Multi-glycoside of Tripterygium wilfordii Hook f. ameliorates proteinuria and acute mesangial injury induced by anti-Thy1.1 monoclonal antibodyYigang Wan, Liubao Gu, Koichi Suzuki, et al.
Free Radical Biology & Medicine|July 25, 2012
NADPH oxidase-mediated upregulation of connexin43 contributes to podocyte injuryQiaojing Yan, Kun Gao, Yuan Chi, et al.
Aging|December 27, 2018
Hyperoside attenuates renal aging and injury induced by D-galactose via inhibiting AMPK-ULK1 signaling-mediated autophagyBuhui Liu, Yue Tu, Weiming He, et al.
Journal of Cellular Biochemistry|June 4, 2009
Downregulation of gap junction expression and function by endoplasmic reticulum stressTao Huang, Yigang Wan, Ying Zhu, et al.
Journal of Ethnopharmacology|July 18, 2024
Supplemented Gegen Qinlian Decoction Formula attenuates podocyte mitochondrial fission and renal fibrosis in diabetic kidney disease by inhibiting TNF-α-mediated necroptosis, compared with empagliflozinYu Wang, Lu Yu, Yajing Li, et al.
American Journal of Translational Research|May 21, 2019
Shenkang injection, a modern preparation of Chinese patent medicine, diminishes tubulointerstitial fibrosis in obstructive nephropathy via targeting pericyte-myofibroblast transitionYinglu Liu, Ge Shi, Hongyun Yee, et al.
Phytomedicine : International Journal of Phytotherapy and Phytopharmacology|February 21, 2019
Huangkui capsule alleviates renal tubular epithelial-mesenchymal transition in diabetic nephropathy via inhibiting NLRP3 inflammasome activation and TLR4/NF-κB signalingWenbei Han, Qian Ma, Yinglu Liu, et al.
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