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Cell Chemical Biology|July 19, 2024
Blocking smoothened cholesterylation: A strategy for overcoming drug resistance in cancerSteven Y Cheng, Ying E ZhangSeminars in Cancer Biology|April 13, 2024
Alternative splicing in EMT and TGF-β signaling during cancer progressionYing E Zhang, Christina H StueltenFrontiers in Cell and Developmental Biology|October 29, 2021
Transforming Growth Factor-β: An Agent of Change in the Tumor MicroenvironmentChristina H Stuelten, Ying E ZhangThe EMBO Journal|July 12, 2002
TGF-beta receptor-activated p38 MAP kinase mediates Smad-independent TGF-beta responsesLi Yu, Mindy C Hébert, Ying E ZhangThe Journal of Biological Chemistry|July 23, 2003
Smurf1 facilitates myogenic differentiation and antagonizes the bone morphogenetic protein-2-induced osteoblast conversion by targeting Smad5 for degradationSai-Xia Ying, Zareena J Hussain, Ying E ZhangThe Journal of Biological Chemistry|October 24, 2020
Phosphorylation of SMURF2 by ATM exerts a negative feedback control of DNA damage responseLiu-Ya Tang, Adam Thomas, Ming Zhou, et al.Oncogene|January 11, 2019
TGF-β-induced alternative splicing of TAK1 promotes EMT and drug resistanceVeenu Tripathi, Jee-Hye Shin, Christina H Stuelten, et al.Cancer Cell|June 13, 2006
Smad3 reduces susceptibility to hepatocarcinoma by sensitizing hepatocytes to apoptosis through downregulation of Bcl-2Yu-An Yang, Gen-Mu Zhang, Lionel Feigenbaum, et al.The Journal of Biological Chemistry|June 14, 2013
Ubiquitination of tumor necrosis factor receptor-associated factor 4 (TRAF4) by Smad ubiquitination regulatory factor 1 (Smurf1) regulates motility of breast epithelial and cancer cellsXiangchun Wang, Chaoyang Jin, Yi Tang, et al.Scientific Reports|November 20, 2019
Integration of TGF-β-induced Smad signaling in the insulin-induced transcriptional response in endothelial cellsErine H Budi, Steven Hoffman, Shaojian Gao, et al.Pageof 5