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Science (New York, N.Y.)|March 1, 2003
Genetics and the specificity of the aging processSiegfried Hekimi, Leonard GuarenteDevelopmental Cell|April 26, 2017
Proteostasis or Aging: Let the CHIPs Fall Where They MayRobyn Branicky, Siegfried HekimiAging Cell|March 30, 2010
Two modes of mitochondrial dysfunction lead independently to lifespan extension in Caenorhabditis elegansWen Yang, Siegfried HekimiPlos Biology|December 15, 2010
A mitochondrial superoxide signal triggers increased longevity in Caenorhabditis elegansWen Yang, Siegfried HekimiCellular and Molecular Life Sciences : CMLS|September 5, 2009
When a theory of aging ages badlyJérôme Lapointe, Siegfried HekimiTrends in Genetics : TIG|August 17, 2006
What keeps C. elegans regular: the genetics of defecationRobyn Branicky, Siegfried HekimiDevelopment (Cambridge, England)|October 21, 2005
Specification of muscle neurotransmitter sensitivity by a Paired-like homeodomain protein in Caenorhabditis elegansRobyn Branicky, Siegfried HekimiThe Journal of Biological Chemistry|July 19, 2008
Early mitochondrial dysfunction in long-lived Mclk1+/- miceJérôme Lapointe, Siegfried HekimiPlos One|November 21, 2012
An enhanced immune response of Mclk1⁺/⁻ mutant mice is associated with partial protection from fibrosis, cancer and the development of biomarkers of agingDantong Wang, Ying Wang, Catherine Argyriou, et al.Pageof 2,150