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Cardiovascular Diabetology|January 9, 2024
The translational potential of miR-26 in atherosclerosis and development of agents for its target genes ACC1/2, COL1A1, CPT1A, FBP1, DGAT2, and SMAD7Wujun Chen, Xiaolin Wu, Jianxia Hu, et al.Circulation Research|February 25, 2018
LncRNA ZFAS1 as a SERCA2a Inhibitor to Cause Intracellular Ca2+ Overload and Contractile Dysfunction in a Mouse Model of Myocardial InfarctionYing Zhang, Lei Jiao, Lihua Sun, et al.Cell Death & Disease|December 11, 2019
lncRNA-ZFAS1 induces mitochondria-mediated apoptosis by causing cytosolic Ca2+ overload in myocardial infarction mice modelLei Jiao, Mengmeng Li, Yingchun Shao, et al.European Journal of Pharmacology|August 31, 2020
GDF11 replenishment protects against hypoxia-mediated apoptosis in cardiomyocytes by regulating autophagyLei Jiao, Yingchun Shao, Qi Yu, et al.Biochimica Et Biophysica Acta. Molecular Cell Research|January 5, 2024
GDF11 mitigates high glucose-induced cardiomyocytes apoptosis by inhibiting the ALKBH5-FOXO3-CDR1as/Hippo signaling pathwayYingchun Shao, Mengmeng Li, Yanying Wang, et al.Vascular Pharmacology|November 9, 2022
NAD+ attenuates cardiac injury after myocardial infarction in diabetic mice through regulating alternative splicing of VEGF in macrophagesLei Jiao, Manyu Gong, Xuewen Yang, et al.Pageof 4