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Plos One|July 11, 2013
Circumventing embryonic lethality with Lcmt1 deficiency: generation of hypomorphic Lcmt1 mice with reduced protein phosphatase 2A methyltransferase expression and defects in insulin signalingKennen B MacKay, Yiping Tu, Stephen G Young, et al.
Nucleus (Austin, Tex.)|September 27, 2023
Revisiting the truncated lamin A produced by a commonly used strain of Lmna knockout miceJoonyoung R Kim, Paul H Kim, Ashley Presnell, et al.
Nucleus (Austin, Tex.)|November 15, 2016
LMNA missense mutations causing familial partial lipodystrophy do not lead to an accumulation of prelamin AYiping Tu, Sofía Sánchez-Iglesias, David Araújo-Vilar, et al.
Proceedings of the National Academy of Sciences of the United States of America|September 19, 2018
Fibroblasts lacking nuclear lamins do not have nuclear blebs or protrusions but nevertheless have frequent nuclear membrane rupturesNatalie Y Chen, Paul Kim, Thomas A Weston, et al.
Proceedings of the National Academy of Sciences of the United States of America|June 23, 2021
Increased expression of LAP2β eliminates nuclear membrane ruptures in nuclear lamin-deficient neurons and fibroblastsNatalie Y Chen, Paul H Kim, Yiping Tu, et al.
Human Molecular Genetics|November 9, 2013
New Lmna knock-in mice provide a molecular mechanism for the 'segmental aging' in Hutchinson-Gilford progeria syndromeHea-Jin Jung, Yiping Tu, Shao H Yang, et al.
Journal of Lipid Research|November 1, 2011
Severe hepatocellular disease in mice lacking one or both CaaX prenyltransferasesShao H Yang, Sandy Y Chang, Yiping Tu, et al.
Proceedings of the National Academy of Sciences of the United States of America|February 11, 2010
Abnormal development of the cerebral cortex and cerebellum in the setting of lamin B2 deficiencyCatherine Coffinier, Sandy Y Chang, Chika Nobumori, et al.
Experimental Dermatology|April 19, 2017
A hypomorphic Egfr allele does not ameliorate the palmoplantar keratoderma caused by SLURP1 deficiencyChristopher M Allan, Deanna Tran, Yiping Tu, et al.
Molecular and Cellular Biology|October 15, 2014
An absence of nuclear lamins in keratinocytes leads to ichthyosis, defective epidermal barrier function, and intrusion of nuclear membranes and endoplasmic reticulum into the nuclear chromatinHea-Jin Jung, Angelica Tatar, Yiping Tu, et al.
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