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Arteriosclerosis, Thrombosis, and Vascular Biology|October 10, 2009
Chylomicronemia elicits atherosclerosis in mice--brief reportMichael M Weinstein, Liya Yin, Yiping Tu, et al.
Molecular Biology of the Cell|October 7, 2011
Deficiencies in lamin B1 and lamin B2 cause neurodevelopmental defects and distinct nuclear shape abnormalities in neuronsCatherine Coffinier, Hea-Jin Jung, Chika Nobumori, et al.
Biochemical and Biophysical Research Communications|September 19, 2018
NanoSIMS imaging reveals unexpected heterogeneity in nutrient uptake by brown adipocytesCuiwen He, Xuchen Hu, Thomas A Weston, et al.
JCI Insight|August 23, 2021
Nuclear membrane ruptures underlie the vascular pathology in a mouse model of Hutchinson-Gilford progeria syndromePaul H Kim, Natalie Y Chen, Patrick J Heizer, et al.
Arteriosclerosis, Thrombosis, and Vascular Biology|September 4, 2010
Cholesterol intake modulates plasma triglyceride levels in glycosylphosphatidylinositol HDL-binding protein 1-deficient miceMichael M Weinstein, Yiping Tu, Anne P Beigneux, et al.
Proceedings of the National Academy of Sciences of the United States of America|December 5, 2019
An absence of lamin B1 in migrating neurons causes nuclear membrane ruptures and cell deathNatalie Y Chen, Ye Yang, Thomas A Weston, et al.
Human Molecular Genetics|February 6, 2015
Mice that express farnesylated versions of prelamin A in neurons develop achalasiaShao H Yang, Shiri Procaccia, Hea-Jin Jung, et al.
Proceedings of the National Academy of Sciences of the United States of America|May 8, 2013
Farnesylation of lamin B1 is important for retention of nuclear chromatin during neuronal migrationHea-Jin Jung, Chika Nobumori, Chris N Goulbourne, et al.
The Journal of Investigative Dermatology|February 7, 2014
Palmoplantar keratoderma along with neuromuscular and metabolic phenotypes in Slurp1-deficient miceOludotun Adeyo, Bernard B Allan, Richard H Barnes, et al.
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