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Journal of Neuroscience Research|November 19, 2016
Genetically modified rodent models of SCA17Yiting Cui, Su Yang, Xiao-Jiang Li, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|August 20, 2017
Synergistic Toxicity of Polyglutamine-Expanded TATA-Binding Protein in Glia and Neuronal Cells: Therapeutic Implications for Spinocerebellar Ataxia 17Yang Yang, Su Yang, Jifeng Guo, et al.Cell Reports|December 6, 2018
Shortening the Half-Life of Cas9 Maintains Its Gene Editing Ability and Reduces Neuronal ToxicitySu Yang, Shihua Li, Xiao-Jiang LiFrontiers in Physiology|December 18, 2018
MANF: A New Player in the Control of Energy Homeostasis, and BeyondSu Yang, Shihua Li, Xiao-Jiang LiRare Diseases (Austin, Tex.)|December 30, 2016
Molecular mechanisms underlying Spinocerebellar Ataxia 17 (SCA17) pathogenesisSu Yang, Xiao-Jiang Li, Shihua LiNeuroscience Bulletin|May 24, 2022
CRISPR-Based Genome-Editing Tools for Huntington's Disease Research and TherapyYiyang Qin, Shihua Li, Xiao-Jiang Li, et al.Molecular Neurodegeneration|January 31, 2018
Piperine ameliorates SCA17 neuropathology by reducing ER stressJifeng Guo, Yiting Cui, Qiong Liu, et al.Brain : a Journal of Neurology|June 28, 2011
Neuronal expression of TATA box-binding protein containing expanded polyglutamine in knock-in mice reduces chaperone protein response by impairing the function of nuclear factor-Y transcription factorShanshan Huang, Joseph J Ling, Su Yang, et al.Neuron|January 28, 2014
Age-dependent decrease in chaperone activity impairs MANF expression, leading to Purkinje cell degeneration in inducible SCA17 miceSu Yang, Shanshan Huang, Marta A Gaertig, et al.Journal of Genetics and Genomics = Yi Chuan Xue Bao|July 4, 2012
Influence of species differences on the neuropathology of transgenic Huntington's disease animal modelsXiao-Jiang Li, Shihua LiPageof 110