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Acta Physiologica (Oxford, England)|July 4, 2026
Expression Landscape and Circadian Regulation of lncRNAs in the KidneyLeonore Wigger, Fanny Durussel, Muriel Auberson, et al.The Biochemical Journal|October 5, 2020
Chemically based transmissible ER stress protocols are unsuitable to study cell-to-cell UPR transmissionYohan Bignon, Virginie Poindessous, Luca Rampoldi, et al.Journal of Insect Physiology|July 27, 2015
Rhodnius prolixus smells repellents: Behavioural evidence and test of present and potential compounds inducing repellency in Chagas disease vectorsPaula F Zermoglio, Hadrien Martin-Herrou, Yohan Bignon, et al.Experimental Physiology|July 22, 2025
Empagliflozin does not prevent progression of Dent's disease type 1 in a mouse modelElise de Combiens, Nadia Frachon, Yohan Bignon, et al.Human Mutation|February 18, 2021
Diversity of functional alterations of the ClC-5 exchanger in the region of the proton glutamate in patients with Dent disease 1Imène Sakhi, Yohan Bignon, Nadia Frachon, et al.Kidney International|December 25, 2019
Defective bicarbonate reabsorption in Kir4.2 potassium channel deficient mice impairs acid-base balance and ammonia excretionYohan Bignon, Laurent Pinelli, Nadia Frachon, et al.Cell Death & Disease|April 19, 2020
The cellular prion protein is a stress protein secreted by renal tubular cells and a urinary marker of kidney injuryYohan Bignon, Virginie Poindessous, Hélène Lazareth, et al.Human Mutation|December 6, 2019
Analysis of CLCNKB mutations at dimer-interface, calcium-binding site, and pore reveals a variety of functional alterations in ClC-Kb channel leading to Bartter syndromeYohan Bignon, Imene Sakhi, Sara Bitam, et al.Human Mutation|May 24, 2018
A novel CLCN5 pathogenic mutation supports Dent disease with normal endosomal acidificationYohan Bignon, Alexi Alekov, Nadia Frachon, et al.JCI Insight|February 22, 2022
Renal tubular peroxisomes are dispensable for normal kidney functionCamille Ansermet, Gabriel Centeno, Sylvain Pradervand, et al.Pageof 2