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International Journal of Molecular Sciences|July 9, 2022
Therapeutic Effects of Quetiapine and 5-HT<sub>1A</sub> Receptor Agonism on Hyperactivity in Dopamine-Deficient MiceYukiko Ochiai, Masayo Fujita, Yoko Hagino, et al.Neuropsychopharmacology Reports|May 19, 2022
Interaction between social behavior and paternal age in offspring of the same paternal miceMiho Tanaka, Atsushi Sato, Hiroko Kotajima-Murakami, et al.Molecular Brain|October 18, 2017
Light/dark phase-dependent spontaneous activity is maintained in dopamine-deficient miceMasayo Fujita, Yoko Hagino, Taishi Takeda, et al.Human Genomics|February 3, 2023
Tsc2 mutation rather than Tsc1 mutation dominantly causes a social deficit in a mouse model of tuberous sclerosis complexHirofumi Kashii, Shinya Kasai, Atsushi Sato, et al.Neuropharmacology|August 3, 2016
Loss of GluN2D subunit results in social recognition deficit, social stress, 5-HT<sub>2C</sub> receptor dysfunction, and anhedonia in miceHideko Yamamoto, Etsuko Kamegaya, Yoko Hagino, et al.Behavioural Pharmacology|September 5, 2023
Heterozygous and homozygous gene knockout of the 5-HT1B receptor have different effects on methamphetamine-induced behavioral sensitizationYuki Moriya, Yoshiyuki Kasahara, Kana Ishihara, et al.Molecular Brain|January 10, 2019
Effects of rapamycin on social interaction deficits and gene expression in mice exposed to valproic acid in uteroHiroko Kotajima-Murakami, Toshiyuki Kobayashi, Hirofumi Kashii, et al.Neuropsychopharmacology : Official Publication of the American College of Neuropsychopharmacology|November 5, 2014
Involvement of cholinergic system in hyperactivity in dopamine-deficient miceYoko Hagino, Shinya Kasai, Masayo Fujita, et al.Molecular Brain|December 17, 2013
Involvement of the N-methyl-D-aspartate receptor GluN2D subunit in phencyclidine-induced motor impairment, gene expression, and increased Fos immunoreactivityHideko Yamamoto, Etsuko Kamegaya, Wakako Sawada, et al.Frontiers in Psychiatry|February 21, 2022
Exposure to GABA<sub>A</sub> Receptor Antagonist Picrotoxin in Pregnant Mice Causes Autism-Like Behaviors and Aberrant Gene Expression in OffspringHiroko Kotajima-Murakami, Hideo Hagihara, Atsushi Sato, et al.Pageof 3