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Frontiers in Cellular Neuroscience|June 26, 2020
Huntingtin-Associated Protein 1 in Mouse Hypothalamus Stabilizes Glucocorticoid Receptor in Stress ResponseXingxing Chen, Ning Xin, Yongcheng Pan, et al.EMBO Reports|April 10, 2020
Phosphorylation of myelin regulatory factor by PRKG2 mediates demyelination in Huntington's diseasePeng Yin, Qiong Liu, Yongcheng Pan, et al.Nature Communications|February 29, 2020
Cerebellum-enriched protein INPP5A contributes to selective neuropathology in mouse model of spinocerebellar ataxias type 17Qiong Liu, Shanshan Huang, Peng Yin, et al.Cell & Bioscience|February 24, 2026
NOTCH2NLC GGC repeat expansions cause retinal neurodegeneration in neuronal intranuclear inclusion disease mouse modelHaibo Li, Ying Jiang, Lusi Zhang, et al.Cell & Bioscience|October 7, 2025
HnRNP M expression rescues neurodegeneration in neuronal intranuclear inclusion disease mouse model by restoring dysregulated RNA splicing and transcriptionYongcheng Pan, Yangping Li, Ying Jiang, et al.Scientific Reports|October 25, 2016
Insertion of a knockout-first cassette in Ampd1 gene leads to neonatal death by disruption of neighboring genes expressionYongcheng Pan, Lusi Zhang, Qiong Liu, et al.Biochemical and Biophysical Research Communications|December 2, 2019
PRRT2 frameshift mutation reduces its mRNA stability resulting loss of function in paroxysmal kinesigenic dyskinesiaYongcheng Pan, Qiong Liu, Jennifer Zhang, et al.Advanced Science (Weinheim, Baden-Wurttemberg, Germany)|July 20, 2026
PolyG Fibrils Coalesce Into Nuclear Ribbons That Engage Proteostasis Machinery in Neuronal Intranuclear Inclusion DiseaseHui Dong, Yongcheng Pan, Zhiyao Tang, et al.Nature Communications|November 20, 2020
Author Correction: Truncation of mutant huntingtin in knock-in mice demonstrates exon1 huntingtin is a key pathogenic formHuiming Yang, Su Yang, Liang Jing, et al.Nature Communications|May 24, 2020
Truncation of mutant huntingtin in knock-in mice demonstrates exon1 huntingtin is a key pathogenic formHuiming Yang, Su Yang, Liang Jing, et al.Pageof 3