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Blood|November 21, 2013
Loss of Asxl1 leads to myelodysplastic syndrome-like disease in miceJiapeng Wang, Zhaomin Li, Yongzheng He, et al.Journal of Bone and Mineral Research : the Official Journal of the American Society for Bone and Mineral Research|April 29, 2015
Nf1 Haploinsufficiency Alters Myeloid Lineage Commitment and Function, Leading to Deranged Skeletal HomeostasisSteven D Rhodes, Hao Yang, Ruizhi Dong, et al.Plos One|October 8, 2011
The haploinsufficient hematopoietic microenvironment is critical to the pathological fracture repair in murine models of neurofibromatosis type 1Xiaohua Wu, Shi Chen, Yongzheng He, et al.The Journal of Biological Chemistry|May 31, 2020
Genetic disruption of the small GTPase RAC1 prevents plexiform neurofibroma formation in mice with neurofibromatosis type 1Julie A Mund, SuJung Park, Abbi E Smith, et al.Stem Cell Reports|May 31, 2016
Loss of Asxl1 Alters Self-Renewal and Cell Fate of Bone Marrow Stromal Cell, Leading to Bohring-Opitz-like Syndrome in MicePeng Zhang, Caihong Xing, Steven D Rhodes, et al.Journal of Bone and Mineral Research : the Official Journal of the American Society for Bone and Mineral Research|May 25, 2013
Hyperactive transforming growth factor-β1 signaling potentiates skeletal defects in a neurofibromatosis type 1 mouse modelSteven D Rhodes, Xiaohua Wu, Yongzheng He, et al.Infectious Diseases and Therapy|July 21, 2024
RSV Risk Profile in Hospitalized Adults and Comparison with Influenza and COVID-19 Controls in Valladolid, Spain, 2010-2022Mariana Haeberer, Martin Mengel, Rong Fan, et al.Infectious Diseases and Therapy|October 12, 2024
Respiratory Syncytial Virus Risk Profile in Hospitalized Infants and Comparison with Influenza and COVID-19 Controls in Valladolid, Spain, 2010-2022Mariana Haeberer, Martin Mengel, Rong Fan, et al.Endocrinology|June 23, 2017
Improving Combination Osteoporosis Therapy in a Preclinical Model of Heightened OsteoanabolismYu Shao, Selene Hernandez-Buquer, Paul Childress, et al.Science Advances|January 25, 2017
ASXL1 interacts with the cohesin complex to maintain chromatid separation and gene expression for normal hematopoiesisZhaomin Li, Peng Zhang, Aimin Yan, et al.Pageof 4