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JBMR Plus|January 12, 2026
Transient neonatal hyperparathyroidism caused by a monoallelic <i>TRPV6</i> dominant negative variantJason Lam Shang Leen, Yoshiro Suzuki, Ai Ling Koh, et al.
EMBO Reports|February 17, 2016
Lack of TRPV2 impairs thermogenesis in mouse brown adipose tissueWuping Sun, Kunitoshi Uchida, Yoshiro Suzuki, et al.
Biochemical and Biophysical Research Communications|March 5, 2023
Roles of TRPM4 in immune responses in keratinocytes and identification of a novel TRPM4-activating agentKaori Otsuka Saito, Fumitaka Fujita, Manami Toriyama, et al.
Pflugers Archiv : European Journal of Physiology|April 16, 2004
Mutational and functional analysis of SLC4A4 in a patient with proximal renal tubular acidosisJun Inatomi, Shoko Horita, Nancy Braverman, et al.
Cells|February 15, 2022
Fine-Tuning of Piezo1 Expression and Activity Ensures Efficient Myoblast Fusion during Skeletal MyogenesisHuascar Pedro Ortuste Quiroga, Massimo Ganassi, Shingo Yokoyama, et al.
Proceedings of the National Academy of Sciences of the United States of America|April 24, 2021
A unique mode of keratinocyte death requires intracellular acidificationTakeshi Matsui, Nanako Kadono-Maekubo, Yoshiro Suzuki, et al.
Journal of Bone and Mineral Research : the Official Journal of the American Society for Bone and Mineral Research|November 30, 2006
Marked disturbance of calcium homeostasis in mice with targeted disruption of the Trpv6 calcium channel geneSuzy D C Bianco, Ji-Bin Peng, Hitomi Takanaga, et al.
American Journal of Human Genetics|June 5, 2018
TRPV6 Variants Interfere with Maternal-Fetal Calcium Transport through the Placenta and Cause Transient Neonatal HyperparathyroidismYoshiro Suzuki, David Chitayat, Hirotake Sawada, et al.
Laboratory Investigation; a Journal of Technical Methods and Pathology|November 11, 2018
Sensory nerve supports epithelial stem cell function in healing of corneal epithelium in mice: the role of trigeminal nerve transient receptor potential vanilloid 4Yuka Okada, Takayoshi Sumioka, Kana Ichikawa, et al.
American Journal of Human Genetics|October 13, 2006
Mutations in the tight-junction gene claudin 19 (CLDN19) are associated with renal magnesium wasting, renal failure, and severe ocular involvementMartin Konrad, Andre Schaller, Dominik Seelow, et al.
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