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Plos One|September 3, 2011
Isolation and characterization of CvIV4: a pain inducing α-scorpion toxinAshlee H Rowe, Yucheng Xiao, Joseph Scales, et al.
The Journal of Clinical Investigation|May 9, 2024
Peripherally targeted analgesia via AAV-mediated sensory neuron-specific inhibition of multiple pronociceptive sodium channelsSeung Min Shin, Brandon Itson-Zoske, Fan Fan, et al.
The Journal of Biological Chemistry|June 18, 2004
Structure--activity relationships of hainantoxin-IV and structure determination of active and inactive sodium channel blockersDongling Li, Yucheng Xiao, Xia Xu, et al.
Biomedical Optics Express|July 5, 2022
Deep learning for denoising in a Mueller matrix microscopeXiongjie Yang, Qianhao Zhao, Tongyu Huang, et al.
The Journal of Physiology|July 25, 2012
The human Nav1.5 F1486 deletion associated with long QT syndrome leads to impaired sodium channel inactivation and reduced lidocaine sensitivityWeihua Song, Yucheng Xiao, Hanying Chen, et al.
Toxicon : Official Journal of the International Society on Toxinology|July 26, 2016
Characterization of ion channels on subesophageal ganglion neurons from Chinese tarantula Ornithoctonus huwena: Exploring the myth of the spider insensitive to its venomMeichun Deng, Zhaotun Hu, Tianfu Cai, et al.
Neuropharmacology|January 29, 2014
Huwentoxin-XVI, an analgesic, highly reversible mammalian N-type calcium channel antagonist from Chinese tarantula Ornithoctonus huwenaMeichun Deng, Xuan Luo, Yucheng Xiao, et al.
Glia|January 8, 2026
Müller Glial Kir4.1 Channel Dysfunction in APOE4-KI Model of Alzheimer's DiseaseSurabhi D Abhyankar, Yucheng Xiao, Neha Mahajan, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|June 14, 2011
Molecular basis of the tarantula toxin jingzhaotoxin-III (β-TRTX-Cj1α) interacting with voltage sensors in sodium channel subtype Nav1.5Mingqiang Rong, Jinjun Chen, Huai Tao, et al.
Journal of Medicinal Chemistry|July 9, 2014
Substituted N-(biphenyl-4'-yl)methyl (R)-2-acetamido-3-methoxypropionamides: potent anticonvulsants that affect frequency (use) dependence and slow inactivation of sodium channelsHyosung Lee, Ki Duk Park, Robert Torregrosa, et al.
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