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Cellular Immunology|October 28, 2011
Advantage of higher-avidity CTL specific for Tax against human T-lymphotropic virus-1 infected cells and tumorsTakako Kitazono, Takahiro Okazaki, Natsumi Araya, et al.Traffic (Copenhagen, Denmark)|January 10, 2008
A CD63 mutant inhibits T-cell tropic human immunodeficiency virus type 1 entry by disrupting CXCR4 trafficking to the plasma membraneTakeshi Yoshida, Yuji Kawano, Kei Sato, et al.Virology Journal|July 19, 2017
Prevalence of plasma autoantibody against cancer testis antigen NY-ESO-1 in HTLV-1 infected individuals with different clinical statusYasuo Shiohama, Tadasuke Naito, Toshio Matsuzaki, et al.Virology Journal|December 6, 2017
The CC chemokine ligand (CCL) 1, upregulated by the viral transactivator Tax, can be downregulated by minocycline: possible implications for long-term treatment of HTLV-1-associated myelopathy/tropical spastic paraparesisMineki Saito, Hiroe Sejima, Tadasuke Naito, et al.Cancer Science|November 9, 2010
SMYD3 interacts with HTLV-1 Tax and regulates subcellular localization of TaxKeiyu Yamamoto, Takaomi Ishida, Kazumi Nakano, et al.International Journal of Cancer|February 25, 2005
Transcriptional activation of survivin through the NF-kappaB pathway by human T-cell leukemia virus type I taxHirochika Kawakami, Mariko Tomita, Takehiro Matsuda, et al.Human Immunology|January 26, 2011
A recombinant vesicular stomatitis virus encoding HIV-1 receptors and human OX40 ligand efficiently eliminates HIV-1-infected CD4-positive T cells expressing OX40Chikayuki Tsuruno, Kazu Okuma, Yoshiaki Takahashi, et al.Retrovirology|December 16, 2014
Cytotoxic T lymphocyte lysis of HTLV-1 infected cells is limited by weak HBZ protein expression, but non-specifically enhanced on induction of Tax expressionAileen G Rowan, Koichiro Suemori, Hiroshi Fujiwara, et al.Blood|June 5, 2009
HTLV-1-Tax and ICAM-1 act on T-cell signal pathways to polarize the microtubule-organizing center at the virological synapseMohamed Nejmeddine, Veera S Negi, Sohini Mukherjee, et al.Retrovirology|May 22, 2013
Interferon-α (IFN-α) suppresses HTLV-1 gene expression and cell cycling, while IFN-α combined with zidovudine induces p53 signaling and apoptosis in HTLV-1-infected cellsShuichi Kinpara, Mami Kijiyama, Ayako Takamori, et al.Pageof 24