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Methods in Molecular Biology (Clifton, N.J.)|December 30, 2018
Complement Regulation and Immune Evasion by Hepatitis C VirusYoung-Chan Kwon, Ranjit RayHepatology (Baltimore, Md.)|August 14, 2018
Hepatitis C Virus Manipulates Humans as its Favorite Host for a Long-Term RelationshipRatna B Ray, Ranjit RayWorld Journal of Diabetes|February 26, 2014
Hepatitis C virus infection and insulin resistanceSandip K Bose, Ranjit RayPlos One|September 7, 2012
N-terminal region of gelsolin induces apoptosis of activated hepatic stellate cells by a caspase-dependent mechanismBudhaditya Mazumdar, Keith Meyer, Ranjit RayJournal of Virology|June 23, 2021
SARS-CoV-2 Spike Protein Induces Paracrine Senescence and Leukocyte Adhesion in Endothelial CellsKeith Meyer, Tapas Patra, Vijayamahantesh, et al.Cells|March 27, 2019
Strategies to Circumvent Host Innate Immune Response by Hepatitis C VirusTapas Patra, Ratna B Ray, Ranjit RayAdvances in Cancer Research|February 13, 2021
Hepatitis C virus associated hepatocellular carcinomaMousumi Khatun, Ranjit Ray, Ratna B RayViruses|October 14, 2011
Oncogenic potential of hepatitis C virus proteinsArup Banerjee, Ratna B Ray, Ranjit RayVirology|June 23, 2004
Influence of N-linked glycans on intracellular transport of hepatitis C virus E1 chimeric glycoprotein and its role in pseudotype virus infectivityAster Beyene, Arnab Basu, Keith Meyer, et al.Journal of Virology|April 14, 2004
The hypervariable region 1 of the E2 glycoprotein of hepatitis C virus binds to glycosaminoglycans, but this binding does not lead to infection in a pseudotype systemArnab Basu, Aster Beyene, Keith Meyer, et al.Pageof 18