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Human Reproduction (Oxford, England)|December 16, 2010
Thrombin-induced chemokine production in endometrial stromal cellsYasushi Kawano, Yuichi Furukawa, Yukie Kawano, et al.
Gynecologic and Obstetric Investigation|October 14, 2004
Adenoid cystic carcinoma of Bartholin's Gland. Case report with review of the literatureKaei Nasu, Yukie Kawano, Noriyuki Takai, et al.
Gynecologic and Obstetric Investigation|December 17, 2010
Cadmium chloride induces the expression of metallothionein mRNA by endometrial stromal cells and amnion-derived (WISH) cellsYasushi Kawano, Yuichi Furukawa, Yukie Kawano, et al.
Human Reproduction (Oxford, England)|January 8, 2013
miR-196b targets c-myc and Bcl-2 expression, inhibits proliferation and induces apoptosis in endometriotic stromal cellsWakana Abe, Kaei Nasu, Chisato Nakada, et al.
European Journal of Obstetrics, Gynecology, and Reproductive Biology|November 30, 2010
Attachment to extracellular matrices is enhanced in human endometriotic stromal cells: a possible mechanism underlying the pathogenesis of endometriosisMasatake Adachi, Kaei Nasu, Akitoshi Tsuno, et al.
Frontiers in Bioscience (Landmark Edition)|June 5, 2014
Aberrant histone modification in endometriosisKaei Nasu, Yukie Kawano, Kentaro Kai, et al.
The Journal of Clinical Endocrinology and Metabolism|September 16, 2011
Fasudil inhibits the proliferation and contractility and induces cell cycle arrest and apoptosis of human endometriotic stromal cells: a promising agent for the treatment of endometriosisAkitoshi Tsuno, Kaei Nasu, Yukie Kawano, et al.
Human Reproduction (Oxford, England)|July 1, 2011
Application of the histone deacetylase inhibitors for the treatment of endometriosis: histone modifications as pathogenesis and novel therapeutic targetYukie Kawano, Kaei Nasu, Haili Li, et al.
Reproductive Sciences (Thousand Oaks, Calif.)|January 4, 2011
Aberrant expression of apoptosis-related molecules in endometriosis: a possible mechanism underlying the pathogenesis of endometriosisKaei Nasu, Masakazu Nishida, Yukie Kawano, et al.
The Journal of Clinical Endocrinology and Metabolism|July 9, 2013
CCAAT/enhancer-binding protein α is epigenetically silenced by histone deacetylation in endometriosis and promotes the pathogenesis of endometriosisYukie Kawano, Kaei Nasu, Naoki Hijiya, et al.
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