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The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|September 24, 2004
Phosphatidylinositol 3-kinase activates ERK in primary sensory neurons and mediates inflammatory heat hyperalgesia through TRPV1 sensitizationZhi-Ye Zhuang, Haoxing Xu, David E Clapham, et al.
The European Journal of Neuroscience|July 6, 2004
Peripheral noxious stimulation induces phosphorylation of the NMDA receptor NR1 subunit at the PKC-dependent site, serine-896, in spinal cord dorsal horn neuronsGary J Brenner, Ru-Rong Ji, Sebastian Shaffer, et al.
The Neuroscientist : a Review Journal Bringing Neurobiology, Neurology and Psychiatry|June 29, 2010
Glial cells and chronic painRomain-Daniel Gosselin, Marc R Suter, Ru-Rong Ji, et al.
Frontiers in Immunology|November 22, 2021
Interferons in Pain and Infections: Emerging Roles in Neuro-Immune and Neuro-Glial InteractionsPing-Heng Tan, Jasmine Ji, Chun-Chang Yeh, et al.
Neuroscience Bulletin|April 20, 2021
Emerging Role of PD-1 in the Central Nervous System and Brain DiseasesJunli Zhao, Alexus Roberts, Zilong Wang, et al.
Expert Opinion on Therapeutic Targets|August 14, 2023
Targeting dorsal root ganglia and primary sensory neurons for the treatment of chronic pain: an updateTemugin Berta, Judith A Strong, Jun-Ming Zhang, et al.
JCI Insight|October 18, 2022
Hevin/Sparcl1 drives pathological pain through spinal cord astrocyte and NMDA receptor signalingGang Chen, Jing Xu, Hao Luo, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|May 24, 2003
p38 mitogen-activated protein kinase is activated after a spinal nerve ligation in spinal cord microglia and dorsal root ganglion neurons and contributes to the generation of neuropathic painShan-Xue Jin, Zhi-Ye Zhuang, Clifford J Woolf, et al.
Frontiers in Pharmacology|July 24, 2019
Resolvin D5 Inhibits Neuropathic and Inflammatory Pain in Male But Not Female Mice: Distinct Actions of D-Series Resolvins in Chemotherapy-Induced Peripheral NeuropathyXin Luo, Yun Gu, Xueshu Tao, et al.
Pain|April 26, 2002
The pattern of expression of the voltage-gated sodium channels Na(v)1.8 and Na(v)1.9 does not change in uninjured primary sensory neurons in experimental neuropathic pain modelsIsabelle Decosterd, Ru-Rong Ji, Salahadin Abdi, et al.
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