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Antioxidants & Redox Signaling|February 14, 2020
Central and Peripheral Metabolic Defects Contribute to the Pathogenesis of Alzheimer's Disease: Targeting Mitochondria for Diagnosis and PreventionYunhua Peng, Peipei Gao, Le Shi, et al.
Acta Biochimica Polonica|April 4, 2020
MiR-98-5p expression inhibits polarization of macrophages to an M2 phenotype by targeting Trib1 in inflammatory bowel diseaseYunhua Peng, Qingyuan Wang, Wei Yang, et al.
Free Radical Research|April 24, 2018
Hydrogen-rich water improves cognitive impairment gender-dependently in APP/PS1 mice without affecting Aβ clearanceChen Hou, Yunhua Peng, Chuan Qin, et al.
Chemistry & Biodiversity|January 19, 2012
Antioxidant phenolic compounds from the stems of Entada phaseoloidesYuqiong Dong, Haiming Shi, Haisha Yang, et al.
Journal of Neurochemistry|March 2, 2016
Mitochondrial dysfunction precedes depression of AMPK/AKT signaling in insulin resistance induced by high glucose in primary cortical neuronsYunhua Peng, Jing Liu, Le Shi, et al.
Frontiers in Psychiatry|February 8, 2018
Correlations among Psychological Resilience, Self-Efficacy, and Negative Emotion in Acute Myocardial Infarction Patients after Percutaneous Coronary InterventionNeng Liu, Shaohui Liu, Nan Yu, et al.
Journal of Neurochemistry|November 19, 2015
Early inflammation-associated factors blunt sterol regulatory element-binding proteins-1-mediated lipogenesis in high-fat diet-fed APPSWE /PSEN1dE9 mouse model of Alzheimer's diseaseYing Tang, Yunhua Peng, Jing Liu, et al.
International Journal of Clinical Practice|April 1, 2021
Level of phosphate diets effect on fibroblast growth factor-23 levels in chronic kidney disease subjects: A meta-analysisZhong Li, Maorong Hu, Yinxue Guo, et al.
Neurobiology of Aging|May 23, 2017
Early interleukin-6 enhances hepatic ketogenesis in APPSWE/PSEN1dE9 mice via 3-hydroxy-3-methylglutary-CoA synthase 2 signaling activation by p38/nuclear factor κB p65Le Shi, Daina Zhao, Chen Hou, et al.
Free Radical Biology & Medicine|January 15, 2014
Reloading functionally ameliorates disuse-induced muscle atrophy by reversing mitochondrial dysfunction, and similar benefits are gained by administering a combination of mitochondrial nutrientsJing Liu, Yunhua Peng, Zhihui Feng, et al.
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