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Biological & Pharmaceutical Bulletin|January 10, 2018
Transcriptome Analysis Reveals That Midnolin Regulates mRNA Expression Levels of Multiple Parkinson's Disease Causative GenesYutaro Obara, Kuniaki IshiiJournal of Pharmacological Sciences|November 21, 2012
Endocytic regulation of voltage-dependent potassium channels in the heartKuniaki Ishii, Ikuo Norota, Yutaro ObaraBiological & Pharmaceutical Bulletin|March 1, 2023
Structural Variants of Midnolin, a Genetic Risk Factor for Parkinson's Disease, in a Yamagata CohortHidenori Sato, Kuniaki Ishii, Yutaro ObaraPflugers Archiv : European Journal of Physiology|September 27, 2013
Insulin suppresses IKs (KCNQ1/KCNE1) currents, which require β-subunit KCNE1Minghua Wu, Yutaro Obara, Ikuo Norota, et al.Annals of Clinical and Translational Neurology|October 8, 2019
Midnolin is a confirmed genetic risk factor for Parkinson's diseaseYutaro Obara, Hidenori Sato, Takahiro Nakayama, et al.Biochemical and Biophysical Research Communications|September 9, 2017
Insulin treatment augments KCNQ1/KCNE1 currents but not KCNQ1 currents, which is associated with an increase in KCNE1 expressionMinghua Wu, Yutaro Obara, Shingo Ohshima, et al.The Journal of Biological Chemistry|September 12, 2019
Molecular identification of HSPA8 as an accessory protein of a hyperpolarization-activated chloride channel from rat pulmonary vein cardiomyocytesYosuke Okamoto, Yoshinobu Nagasawa, Yutaro Obara, et al.The Journal of Pharmacology and Experimental Therapeutics|March 4, 2022
Insulin Enhances Gene Expression of Midnolin, a Novel Genetic Risk Factor for Parkinson's Disease, via Extracellular Signal-Regulated Kinase, Phosphoinositide 3-Kinase and Multiple Transcription Factors in SH-SY5Y CellsNaoki Sagehashi, Yutaro Obara, Ohki Maruyama, et al.International Journal of Molecular Sciences|July 13, 2018
ERK5 Phosphorylates Kv4.2 and Inhibits Inactivation of the A-Type Current in PC12 CellsYurina Kashino, Yutaro Obara, Yosuke Okamoto, et al.Scientific Reports|July 21, 2017
Midnolin is a novel regulator of parkin expression and is associated with Parkinson's DiseaseYutaro Obara, Toru Imai, Hidenori Sato, et al.Pageof 8