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Z F Ba

Showing results (1-10 of 61) with videos related to

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The American Journal of Physiology|December 1, 1992
Differential effects of ATP-MgCl2 on portal and hepatic arterial blood flow after hemorrhage and resuscitationP Wang, Z F Ba, I H Chaudry
Archives of Surgery (Chicago, Ill. : 1960)|April 1, 1997
Mechanism of hepatocellular dysfunction during early sepsis. Key role of increased gene expression and release of proinflammatory cytokines tumor necrosis factor and interleukin-6P Wang, Z F Ba, I H Chaudry
The American Journal of Physiology|June 1, 1994
Administration of tumor necrosis factor-alpha in vivo depresses endothelium-dependent relaxationP Wang, Z F Ba, I H Chaudry
Archives of Surgery (Chicago, Ill. : 1960)|November 1, 1994
Nitric oxide. To block or enhance its production during sepsis?P Wang, Z F Ba, I H Chaudry
Circulatory Shock|April 1, 1992
ATP-MgCl2 restores the depressed cardiac output following trauma and severe hemorrhage even in the absence of blood resuscitationP Wang, Z F Ba, I H Chaudry
The American Journal of Physiology|November 1, 1995
Endothelium-dependent relaxation is depressed at the macro- and microcirculatory levels during sepsisP Wang, Z F Ba, I H Chaudry
The American Journal of Physiology|April 1, 1995
ATP-MgCl2 restores depressed endothelial cell function after hemorrhagic shock and resuscitationP Wang, Z F Ba, I H Chaudry
Archives of Surgery (Chicago, Ill. : 1960)|February 1, 1991
Hepatic extraction of indocyanine green is depressed early in sepsis despite increased hepatic blood flow and cardiac outputP Wang, Z F Ba, I H Chaudry
Shock (Augusta, Ga.)|January 1, 1995
Hepatocellular dysfunction occurs earlier than the onset of hyperdynamic circulation during sepsisP Wang, Z F Ba, I H Chaudry
The American Journal of Physiology|December 1, 1991
Increase in hepatic blood flow during early sepsis is due to increased portal blood flowP Wang, Z F Ba, I H Chaudry
Pageof 7

Showing results (1-10 of 61) with videos related to

Sort By:
Pageof 7
The American Journal of Physiology|December 1, 1992
Differential effects of ATP-MgCl2 on portal and hepatic arterial blood flow after hemorrhage and resuscitationP Wang, Z F Ba, I H Chaudry
Archives of Surgery (Chicago, Ill. : 1960)|April 1, 1997
Mechanism of hepatocellular dysfunction during early sepsis. Key role of increased gene expression and release of proinflammatory cytokines tumor necrosis factor and interleukin-6P Wang, Z F Ba, I H Chaudry
The American Journal of Physiology|June 1, 1994
Administration of tumor necrosis factor-alpha in vivo depresses endothelium-dependent relaxationP Wang, Z F Ba, I H Chaudry
Archives of Surgery (Chicago, Ill. : 1960)|November 1, 1994
Nitric oxide. To block or enhance its production during sepsis?P Wang, Z F Ba, I H Chaudry
Circulatory Shock|April 1, 1992
ATP-MgCl2 restores the depressed cardiac output following trauma and severe hemorrhage even in the absence of blood resuscitationP Wang, Z F Ba, I H Chaudry
The American Journal of Physiology|November 1, 1995
Endothelium-dependent relaxation is depressed at the macro- and microcirculatory levels during sepsisP Wang, Z F Ba, I H Chaudry
The American Journal of Physiology|April 1, 1995
ATP-MgCl2 restores depressed endothelial cell function after hemorrhagic shock and resuscitationP Wang, Z F Ba, I H Chaudry
Archives of Surgery (Chicago, Ill. : 1960)|February 1, 1991
Hepatic extraction of indocyanine green is depressed early in sepsis despite increased hepatic blood flow and cardiac outputP Wang, Z F Ba, I H Chaudry
Shock (Augusta, Ga.)|January 1, 1995
Hepatocellular dysfunction occurs earlier than the onset of hyperdynamic circulation during sepsisP Wang, Z F Ba, I H Chaudry
The American Journal of Physiology|December 1, 1991
Increase in hepatic blood flow during early sepsis is due to increased portal blood flowP Wang, Z F Ba, I H Chaudry
Pageof 7