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Molecular Cell|April 8, 2004
Eukaryotic proteasomes cannot digest polyglutamine sequences and release them during degradation of polyglutamine-containing proteinsPrasanna Venkatraman, Ronald Wetzel, Motomasa Tanaka, et al.
Biochemistry|February 3, 2009
Isolation of mammalian 26S proteasomes and p97/VCP complexes using the ubiquitin-like domain from HHR23B reveals novel proteasome-associated proteinsHenrike C Besche, Wilhelm Haas, Steven P Gygi, et al.
Frontiers in Molecular Neuroscience|August 19, 2010
Characterization of the Brain 26S Proteasome and its Interacting ProteinsHwan-Ching Tai, Henrike Besche, Alfred L Goldberg, et al.
Advances in Immunology|June 25, 2002
Protein degradation and the generation of MHC class I-presented peptidesKenneth L Rock, Ian A York, Tomo Saric, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|November 1, 2002
Patterns of gene expression in atrophying skeletal muscles: response to food deprivationR Thomas Jagoe, Stewart H Lecker, Marcelo Gomes, et al.
The Journal of Biological Chemistry|March 28, 2012
Cathepsins L and Z are critical in degrading polyglutamine-containing proteins within lysosomesNidhi Bhutani, Rosanna Piccirillo, Raphael Hourez, et al.
Proceedings of the National Academy of Sciences of the United States of America|February 21, 2019
26S Proteasomes are rapidly activated by diverse hormones and physiological states that raise cAMP and cause Rpn6 phosphorylationJordan J S VerPlank, Sudarsanareddy Lokireddy, Jinghui Zhao, et al.
The International Journal of Biochemistry & Cell Biology|June 1, 2013
Myostatin/activin pathway antagonism: molecular basis and therapeutic potentialH Q Han, Xiaolan Zhou, William E Mitch, et al.
Molecular Cell|January 22, 2003
ATP hydrolysis by the proteasome regulatory complex PAN serves multiple functions in protein degradationNadia Benaroudj, Peter Zwickl, Erika Seemüller, et al.
American Journal of Physiology. Endocrinology and Metabolism|April 22, 2004
IGF-I stimulates muscle growth by suppressing protein breakdown and expression of atrophy-related ubiquitin ligases, atrogin-1 and MuRF1Jennifer M Sacheck, Akira Ohtsuka, S Christine McLary, et al.
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