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Molecular Pharmaceutics|August 3, 2012
Sequence-dependent synergistic inhibition of human glioma cell lines by combined temozolomide and miR-21 inhibitor gene therapyXiaomin Qian, Yu Ren, Zhendong Shi, et al.
World Journal of Surgery|May 27, 2026
Dynamic Evolution of HER2 Expression Following Neoadjuvant Therapy and Its Association With Treatment Response and Prognosis in Breast Cancer: A Large-Scale Retrospective StudyShunan Wang, Tong Liu, Xu Liu, et al.
International Journal of Oncology|July 2, 2011
β-catenin/Tcf-4 complex transcriptionally regulates AKT1 in gliomaLingchao Chen, Kai Huang, Lei Han, et al.
Cancer Letters|January 18, 2015
DNMT1 and EZH2 mediated methylation silences the microRNA-200b/a/429 gene and promotes tumor progressionXianghong Ning, Zhendong Shi, Xi Liu, et al.
Frontiers in Oncology|January 28, 2026
Correction: Chidamide combined with doxorubicin induced p53-driven cell cycle arrest and cell apoptosis reverse multidrug resistance of breast cancerLixia Cao, Shaorong Zhao, Qianxi Yang, et al.
Journal of Experimental & Clinical Cancer Research : CR|April 7, 2021
Chemotherapy-elicited exosomal miR-378a-3p and miR-378d promote breast cancer stemness and chemoresistance via the activation of EZH2/STAT3 signalingQianxi Yang, Shaorong Zhao, Zhendong Shi, et al.
Frontiers in Oncology|April 9, 2021
Over-Expression of Centromere Protein U Participates in the Malignant Neoplastic Progression of Breast CancerXiaomeng Hao, Yufan Qiu, Lixia Cao, et al.
International Journal of Oncology|January 3, 2012
MicroRNA-200a suppresses the Wnt/β-catenin signaling pathway by interacting with β-cateninJuan Su, Anling Zhang, Zhendong Shi, et al.
American Journal of Cancer Research|December 15, 2025
Erratum: Deciphering the performance of polo-like kinase 1 in triple-negative breast cancer progression according to the centromere protein U-phosphorylation pathwayShaorong Zhao, Yannan Geng, Lixia Cao, et al.
Cancer Letters|November 10, 2019
Centromere protein U (CENPU) enhances angiogenesis in triple-negative breast cancer by inhibiting ubiquitin-proteasomal degradation of COX-2Teng Pan, Dongdong Zhou, Zhendong Shi, et al.
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