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Molecular and Cellular Biology
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June 22, 2002
A two-stage, p16(INK4A)- and p53-dependent keratinocyte senescence mechanism that limits replicative potential independent of telomere status
James G Rheinwald, William C Hahn, Matthew R Ramsey, et al.
Scientific Reports
|
March 7, 2020
Recapitulating T cell infiltration in 3D psoriatic skin models for patient-specific drug testing
Jung U Shin, Hasan E Abaci, Lauren Herron, et al.
Cell Cycle (Georgetown, Tex.)
|
July 18, 2012
Tetraploid cells from cytokinesis failure induce aneuploidy and spontaneous transformation of mouse ovarian surface epithelial cells
Lei Lv, Tianwei Zhang, Qiyi Yi, et al.
Science Translational Medicine
|
November 28, 2014
Induced pluripotent stem cells from human revertant keratinocytes for the treatment of epidermolysis bullosa
Noriko Umegaki-Arao, Anna M G Pasmooij, Munenari Itoh, et al.
Proceedings of the National Academy of Sciences of the United States of America
|
December 11, 2019
CRISPR/Cas9-based targeted genome editing for correction of recessive dystrophic epidermolysis bullosa using iPS cells
Joanna Jacków, Zongyou Guo, Corey Hansen, et al.
Bioengineering & Translational Medicine
|
February 3, 2022
Engineering human skin model innervated with itch sensory neuron-like cells differentiated from induced pluripotent stem cells
Zongyou Guo, Chi-Kun Tong, Joanna Jacków, et al.
Nature Biomedical Engineering
|
April 28, 2022
A multi-organ chip with matured tissue niches linked by vascular flow
Kacey Ronaldson-Bouchard, Diogo Teles, Keith Yeager, et al.
Biorxiv : the Preprint Server for Biology
|
March 13, 2023
A scalable, GMP-compatible, autologous organotypic cell therapy for Dystrophic Epidermolysis Bullosa
Gernot Neumayer, Jessica L Torkelson, Shengdi Li, et al.
Nature Communications
|
July 11, 2024
A scalable and cGMP-compatible autologous organotypic cell therapy for Dystrophic Epidermolysis Bullosa
Gernot Neumayer, Jessica L Torkelson, Shengdi Li, et al.
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Search research articles
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Showing results (11-20 of 19) with videos related to
Sort By:
Page
of 2
You have reached the last page of results.
This site can display upto 19 results.
Molecular and Cellular Biology
|
June 22, 2002
A two-stage, p16(INK4A)- and p53-dependent keratinocyte senescence mechanism that limits replicative potential independent of telomere status
James G Rheinwald, William C Hahn, Matthew R Ramsey, et al.
Scientific Reports
|
March 7, 2020
Recapitulating T cell infiltration in 3D psoriatic skin models for patient-specific drug testing
Jung U Shin, Hasan E Abaci, Lauren Herron, et al.
Cell Cycle (Georgetown, Tex.)
|
July 18, 2012
Tetraploid cells from cytokinesis failure induce aneuploidy and spontaneous transformation of mouse ovarian surface epithelial cells
Lei Lv, Tianwei Zhang, Qiyi Yi, et al.
Science Translational Medicine
|
November 28, 2014
Induced pluripotent stem cells from human revertant keratinocytes for the treatment of epidermolysis bullosa
Noriko Umegaki-Arao, Anna M G Pasmooij, Munenari Itoh, et al.
Proceedings of the National Academy of Sciences of the United States of America
|
December 11, 2019
CRISPR/Cas9-based targeted genome editing for correction of recessive dystrophic epidermolysis bullosa using iPS cells
Joanna Jacków, Zongyou Guo, Corey Hansen, et al.
Bioengineering & Translational Medicine
|
February 3, 2022
Engineering human skin model innervated with itch sensory neuron-like cells differentiated from induced pluripotent stem cells
Zongyou Guo, Chi-Kun Tong, Joanna Jacków, et al.
Nature Biomedical Engineering
|
April 28, 2022
A multi-organ chip with matured tissue niches linked by vascular flow
Kacey Ronaldson-Bouchard, Diogo Teles, Keith Yeager, et al.
Biorxiv : the Preprint Server for Biology
|
March 13, 2023
A scalable, GMP-compatible, autologous organotypic cell therapy for Dystrophic Epidermolysis Bullosa
Gernot Neumayer, Jessica L Torkelson, Shengdi Li, et al.
Nature Communications
|
July 11, 2024
A scalable and cGMP-compatible autologous organotypic cell therapy for Dystrophic Epidermolysis Bullosa
Gernot Neumayer, Jessica L Torkelson, Shengdi Li, et al.
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