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Journal of Biochemical and Molecular Toxicology|November 2, 2018
Bldesin, the first functionally characterized pathogenic fungus defensin with Kv1.3 channel and chymotrypsin inhibitory activitiesXudong Luo, Wen Zhu, Li Ding, et al.Scientific Reports|May 9, 2015
Toxin acidic residue evolutionary function-guided design of de novo peptide drugs for the immunotherapeutic target, the Kv1.3 channelZongyun Chen, Youtian Hu, Jing Hong, et al.Toxicon : Official Journal of the International Society on Toxinology|July 18, 2018
The Kv1.3 channel-inhibitory toxin BF9 also displays anticoagulant activity via inhibition of factor XIaLi Ding, Jinbo Hao, Xudong Luo, et al.Toxins|January 24, 2025
Improvement in XIa Selectivity of Snake Venom Peptide Analogue BF9-N17K Using P2' Amino Acid ReplacementsLi Ding, Zhiping Zhai, Tianxiang Qin, et al.Frontiers in Microbiology|December 17, 2021
Corrigendum: Fine-Tuning of Alkaline Residues on the Hydrophilic Face Provides a Non-toxic Cationic α-Helical Antimicrobial Peptide Against Antibiotic-Resistant ESKAPE PathogensXudong Luo, Xiangdong Ye, Li Ding, et al.Frontiers in Microbiology|August 2, 2021
Fine-Tuning of Alkaline Residues on the Hydrophilic Face Provides a Non-toxic Cationic α-Helical Antimicrobial Peptide Against Antibiotic-Resistant ESKAPE PathogensXudong Luo, Xiangdong Ye, Li Ding, et al.Toxicon : Official Journal of the International Society on Toxinology|January 13, 2017
St20, a new venomous animal derived natural peptide with immunosuppressive and anti-inflammatory activitiesMin Xiao, Li Ding, Weishan Yang, et al.The Journal of Biological Chemistry|November 9, 2018
A scorpion venom peptide Ev37 restricts viral late entry by alkalizing acidic organellesFangfang Li, Yange Lang, Zhenglin Ji, et al.The Journal of Biological Chemistry|March 21, 2013
Two conserved arginine residues from the SK3 potassium channel outer vestibule control selectivity of recognition by scorpion toxinsJing Feng, Youtian Hu, Hong Yi, et al.Journal of Cellular Biochemistry|November 15, 2018
Programmed cell death protein 4 deficiency suppresses foam cell formation by activating autophagy in advanced glycation end-product low-density lipoprotein-induced macrophagesShan Li, Guangdong Gao, Fuyun Wu, et al.Pageof 7