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Birth Defects Research. Part A, Clinical and Molecular Teratology|March 3, 2006
Myo-inositol enhances teratogenicity of valproic acid in the mouseValentina Massa, Bogdan Wlodarczyk, Erminio Giavini, et al.Birth Defects Research. Part A, Clinical and Molecular Teratology|April 1, 2006
Extremely high prevalence of neural tube defects in a 4-county area in Shanxi Province, ChinaZhiwen Li, Aiguo Ren, Le Zhang, et al.Birth Defects Research. Part A, Clinical and Molecular Teratology|August 26, 2006
Maternal birthplace and major congenital malformations among New York HispanicsMotao Zhu, Charlotte Druschel, Shao LinBirth Defects Research. Part A, Clinical and Molecular Teratology|September 7, 2006
Quality and content of abstracts in papers reporting about drug exposures during pregnancyThomas R Einarson, Crystal Lee, Ryan Smith, et al.Birth Defects Research. Part A, Clinical and Molecular Teratology|September 7, 2006
Further evidence of association between mutations in FGFR2 and syndromic craniosynostosis with sacrococcygeal eversionNélio A J Oliveira, Luís G Alonso, Roberto D Fanganiello, et al.Birth Defects Research. Part A, Clinical and Molecular Teratology|September 26, 2006
Wnt9b is the mutated gene involved in multifactorial nonsyndromic cleft lip with or without cleft palate in A/WySn mice, as confirmed by a genetic complementation testDiana M Juriloff, Muriel J Harris, Andrew P McMahon, et al.Birth Defects Research. Part A, Clinical and Molecular Teratology|September 26, 2006
Gestational stage sensitivity to ultrasound effect on postnatal growth and development of miceSuresh Rao, Nikolai Ovchinnikov, Amanda McRaeBirth Defects Research. Part A, Clinical and Molecular Teratology|October 6, 2006
Reduction of all-trans-retinoic acid-induced teratogenesis in the rat by glycine administrationAlba Martínez-Angoa, Elías Parra-Hernández, Eduardo Madrigal-Bujaidar, et al.Birth Defects Research. Part A, Clinical and Molecular Teratology|August 26, 2006
Evidence for a functional genetic polymorphism of the human retinoic acid-metabolizing enzyme CYP26A1, an enzyme that may be involved in spina bifidaEmmanuel Rat, Ingrid Billaut-Laden, Delphine Allorge, et al.Birth Defects Research. Part A, Clinical and Molecular Teratology|August 26, 2006
Teratogen update: pseudoephedrineMartha M WerlerPageof 126