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Circulation Research|October 6, 2007
Targeted deletion of thioredoxin-interacting protein regulates cardiac dysfunction in response to pressure overloadJun Yoshioka, Kenichi Imahashi, Scott A Gabel, et al.Circulation Research|August 2, 2008
Tra2beta as a novel mediator of vascular smooth muscle diversificationSupriya Shukla, Steven A FisherCirculation Research|August 5, 2008
Endothelium-specific interference with peroxisome proliferator activated receptor gamma causes cerebral vascular dysfunction in response to a high-fat dietAndreas M Beyer, Willem J de Lange, Carmen M Halabi, et al.Circulation Research|May 2, 2009
Genetically determined differences in sodium current characteristics modulate conduction disease severity in mice with cardiac sodium channelopathyCarol Ann Remme, Brendon P Scicluna, Arie O Verkerk, et al.Circulation Research|May 16, 2009
Milk fat globule protein epidermal growth factor-8: a pivotal relay element within the angiotensin II and monocyte chemoattractant protein-1 signaling cascade mediating vascular smooth muscle cells invasionZongming Fu, Mingyi Wang, Marjan Gucek, et al.Circulation Research|February 21, 2009
Lipid incorporation inhibits Src-dependent assembly of fibronectin and type I collagen by vascular smooth muscle cellsMatthew J Frontini, Caroline O'Neil, Cynthia Sawyez, et al.Circulation Research|April 18, 2009
3-Deazaadenosine prevents smooth muscle cell proliferation and neointima formation by interfering with Ras signalingDaniel G Sedding, Monique Tröbs, Fabian Reich, et al.Circulation Research|August 9, 2008
A Rho kinase/myocardin-related transcription factor-A-dependent mechanism underlies the sphingosylphosphorylcholine-induced differentiation of mesenchymal stem cells into contractile smooth muscle cellsEun Su Jeon, Won Sun Park, Mi Jeong Lee, et al.Circulation Research|August 9, 2008
Tumor necrosis factor-alpha downregulates endothelial nitric oxide synthase mRNA stability via translation elongation factor 1-alpha 1Guijun Yan, Bei You, Shao-Ping Chen, et al.Circulation Research|October 4, 2008
Ca2+ influx through T- and L-type Ca2+ channels have different effects on myocyte contractility and induce unique cardiac phenotypesNaser Jaleel, Hiroyuki Nakayama, Xiongwen Chen, et al.Pageof 1,227