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Inflammation|November 29, 2018
Degradation Products of Polydopamine Restrained Inflammatory Response of LPS-Stimulated Macrophages Through Mediation TLR-4-MYD88 Dependent Signaling Pathways by AntioxidantLiang Jin, Feng Yuan, Chenxin Chen, et al.
Inflammation|November 30, 2018
Insulin Antagonizes LPS-Induced Inflammatory Responses by Activating SR-A1/ERK Axis in MacrophagesLiu Zhu, Lei Fan, Yaqin Zhu, et al.
Inflammation|January 16, 2019
Umbelliferone Alleviates Lipopolysaccharide-Induced Inflammatory Responses in Acute Lung Injury by Down-Regulating TLR4/MyD88/NF-κB SignalingDongqiu Wang, Xia Wang, Wen Tong, et al.
Inflammation|December 6, 2019
Ellipticine Conveys Protective Effects to Lipopolysaccharide-Activated Macrophages by Targeting the JNK/AP-1 Signaling PathwayLi-Xing Tian, Xiao-Yu Li, Xin Tang, et al.
Inflammation|November 28, 2019
Ar-Turmerone Exerts Anti-proliferative and Anti-inflammatory Activities in HaCaT Keratinocytes by Inactivating Hedgehog PathwaySen Yang, Jia Liu, Jianxia Jiao, et al.
Inflammation|December 4, 2019
Immunostimulatory Activities of Coliphages on In Vitro Activated Mammalian MacrophagesGizem Yıldızlı, Gokhan Coral, Furkan Ayaz
Inflammation|December 18, 2019
Hederacoside-C Inhibition of Staphylococcus aureus-Induced Mastitis via TLR2 & TLR4 and Their Downstream Signaling NF-κB and MAPKs Pathways In Vivo and In VitroMuhammad Akhtar, Aftab Shaukat, Arshad Zahoor, et al.
Inflammation|June 1, 1977
Effect of leukocyte hydrolases on bacteria. IX. The release of lipoteichoic acid from group A streptococci and from Strep. mutans by leukocyte extracts and by lysozyme: relation to tissue damage in inflammatory sitesM N Sela, M Lahav, I Ginsburg
Inflammation|June 1, 1977
Effect of leukocyte hydrolases on bacteria. X. The role played by leukocyte factors, cationic polyelectrolytes, and by membrane-damaging agents in the lysis of Staphylococcus aureus: relation to chronic inflammatory processesM Lahav, I Ginsburg
Inflammation|February 21, 2018
Periodontitis May Restrain the Mandibular Bone Healing via Disturbing Osteogenic and Osteoclastic BalanceRongjing Zhou, Lili Shen, Chengzhe Yang, et al.
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