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Molecular Endocrinology (Baltimore, Md.)|October 2, 2001
New androgen response elements in the murine pem promoter mediate selective transactivationK Barbulescu, C Geserick, I Schüttke, et al.
Molecular Endocrinology (Baltimore, Md.)|May 29, 2001
Ligands specify coactivator nuclear receptor (NR) box affinity for estrogen receptor subtypesK S Bramlett, Y Wu, T P Burris
Molecular Endocrinology (Baltimore, Md.)|May 29, 2001
Disrupted amino- and carboxyl-terminal interactions of the androgen receptor are linked to androgen insensitivityJ Thompson, F Saatcioglu, O A Jänne, et al.
Molecular Endocrinology (Baltimore, Md.)|May 29, 2001
Müllerian inhibiting substance signaling uses a bone morphogenetic protein (BMP)-like pathway mediated by ALK2 and induces SMAD6 expressionT R Clarke, Y Hoshiya, S E Yi, et al.
Molecular Endocrinology (Baltimore, Md.)|December 16, 2000
Activation of peroxisome proliferator-activated receptors (PPARs) by their ligands and protein kinase A activatorsG Lazennec, L Canaple, D Saugy, et al.
Molecular Endocrinology (Baltimore, Md.)|December 16, 2000
ARIP3 (androgen receptor-interacting protein 3) and other PIAS (protein inhibitor of activated STAT) proteins differ in their ability to modulate steroid receptor-dependent transcriptional activationN Kotaja, S Aittomäki, O Silvennoinen, et al.
Molecular Endocrinology (Baltimore, Md.)|December 16, 2000
Temporally distinct and ligand-specific recruitment of nuclear receptor-interacting peptides and cofactors to subnuclear domains containing the estrogen receptorF Schaufele, C Y Chang, W Liu, et al.
Molecular Endocrinology (Baltimore, Md.)|February 7, 2001
New signaling pathways for hormones and cyclic adenosine 3',5'-monophosphate action in endocrine cellsJ S Richards
Molecular Endocrinology (Baltimore, Md.)|February 7, 2001
Glucocorticoid repression of AP-1 is not mediated by competition for nuclear coactivatorsK De Bosscher, W Vanden Berghe, G Haegeman
Molecular Endocrinology (Baltimore, Md.)|February 7, 2001
Human angiotensin II type 1 receptor isoforms encoded by messenger RNA splice variants are functionally distinctM M Martin, B M Willardson, G F Burton, et al.
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